 The syndrome follows an autosomal dominant pattern, meaning that if one of the two available genes carries a mutation the syndrome will result.Currently, no familial histories are known (in other words, there are no reports of cases in which a parent carrying a mutation in their "FGFR2" gene then propagated said mutation to his or her child).Beare–Stevenson cutis gyrata syndrome is so rare that a reliable incidence cannot be established as of yet; fewer than 25 patients with the condition have been reported.