 Upon analysis of the urinary results, researchers discovered the formation of metabolite, "N"-acetyl-"S"-(1-bromo-3-propyl)-cysteine and the decline in the GSH content of the liver of the rats.This led to the assumption that 1,3-dibromopropane could have reacted with GSH after administration and gave rise to 1-bromo-3-propyl-"S"-glutathione, which ultimately form the urinary metabolite.Moreover, due to little radioactivity observed from feces and the confirmation from maintained blood levels of radioactivity proved the occurrence of biliary excretion of sulfur-containing metabolites and enterohepatic cycling.