For example, the fatal Tay-Sachs disease arises as genetic defects leads to no functional hexosaminidase A produced, causing GM2 to accumulate in lysosomes, ultimately the ganglion cells in the nervous system swells enormously, disturbing the normal functions of neurons.
Ultimately the ganglion cells in the nervous system swell enormously, disturbing the normal functions of neurons. For example, the fatal Tay-Sachs disease arises as a genetic defect which leads to no functional hexosaminidase A produced, causing GM2 to accumulate in lysosomes.
For example, the fatal Tay-Sachs disease arises as a genetic defect which leads to no functional hexosaminidase A produced, causing GM2 to accumulate in lysosomes. Ultimately the ganglion cells in the nervous system swell enormously, disturbing the normal functions of neurons.
For example, the fatal Tay-Sachs disease arises as a genetic defect which leads to no functional hexosaminidase A produced, causing GM2 to accumulate in lysosomes. Ultimately the ganglion cells in the nervous system swell enormously, disturbing the normal functions of neurons.