E. Faecalis has been observed to slowly evolve immunity to a drug which targets this organelle by binding to domain V, those drugs are oxazolidinones. A Fungus of the genus Fusidium produce a different drug which targets this structure. The antifungal cycloheximide functions by targeting this structure. One method of targeting this structure does so by preventing the turnover of EF-G. Many drugs which target this structure, including pleuromutilin and macrolides, do so by inhibiting peptidyl transferase. Tetracyclines inhibit this structure by blocking the attachment of aminoacyl tRNA to the A site. Drugs that target this structure usually target either the 50S or 30S subunits. For 10 points, name this structure, which is targeted to inhibit protein synthesis.
E. Faecalis has been observed to slowly evolve immunity to a drug which targets this organelle by binding to domain V, those drugs are oxazolidinones. A Fungus of the genus Fusidium produce a different drug which targets this structure. The antifungal cycloheximide functions by targeting this structure. One method of targeting this structure does so by preventing the turnover of EF-G. Many drugs which target this structure, including pleuromutilin and macrolides, do so by inhibiting peptidyl transferase. Tetracyclines inhibit this structure by blocking the attachment of aminoacyl tRNA to the A site. Drugs that target this structure usually target either the 50S or 30S subunits. For 10 points, name this structure, which is targeted to inhibit protein synthesis.
E. Faecalis has been observed to slowly evolve immunity to a drug which targets this organelle by binding to domain V, those drugs are oxazolidinones.