MRN recruits ataxia telangiÂ·ectasia mutated during this process to facilitate gamma H2AX production. Lig-4 complexes with XRCC4 during a form of this process. 53BP1 is countered by the activity of BRCA1 when this process is performed during S or G2 phase. A protein kinase complex used for this general process is also needed for telomere stability and B and T cell maturation and is localized to specific sites by its Ku70 and Ku80 proteins. A defect in a type of this process causes xeroderma pigmentosum. Pathways in this process include nonhomologous end-joining, which addresses double-strand breaks such as those caused by ionizing radiation. For 10 points, name this process in which lesions 
MRN recruits ataxia telangiÂ·ectasia mutated during this process to facilitate gamma H2AX production. Lig-4 complexes with XRCC4 during a form of this process. 53BP1 is countered by the activity of BRCA1 when this process is performed during S or G2 phase. A protein kinase complex used for this general process is also needed for telomere stability and B and T cell maturation and is localized to specific sites by its Ku70 and Ku80 proteins. A defect in a type of this process causes xeroderma pigmentosum. Pathways in this process include nonhomologous end-joining, which addresses double-strand breaks such as those caused by ionizing radiation. For 10 points, name this process in which lesions on a namesake genetic molecule are patched.
MRN recruits ataxia telangiÂ·ectasia mutated during this process to facilitate gamma H2AX production.