Defects in mannose phosphorylation lead to their failure and I-cell disease, characterized by inclusion bodies similar to those seen in Tay-Sachs disease. In excess, Vitamin A can induce hepatotoxicity by weakening the membrane of these organelles in the liver. This break in their membrane releases hydrolytic enzymes, which cause the death of the cell. FTP, name these orga
Defects in mannose phosphorylation lead to their failure and I-cell disease, characterized by inclusion bodies similar to those seen in Tay-Sachs disease. In excess, Vitamin A can induce hepatotoxicity by weakening the membrane of these organelles in the liver. This break in their membrane releases hydrolytic enzymes, which cause the death of the cell. FTP, name these organelles active during apoptosis, which act as the digestive system of the cell.
Defects in mannose phosphorylation lead to their failure and I-cell disease, characterized by inclusion bodies similar to those seen in Tay-Sachs disease.
Lysosomes