A member of this protein family crystal ized by Kaspar Locher, Al en Lee, and Douglas Rees was the  first member to have its crystal structure determined and is the E. coli protein BtuCD. The glycoprotein  tapasin mediates the interaction between another member of this family and MHC I molecules; that  member is TAP. The B1, C1, and G2 members of this protein family are responsible for the vast majority of  multidrug resistant tumor cel s. Important motifs in this family include the P-loop, Walker B motif, Q-loop,  H-motif, and their namesake signature motif. Mutations in another member of this family that transfers  fatty acids to peroxisomes causes adrenoleukodystr
A member of this protein family crystal ized by Kaspar Locher, Al en Lee, and Douglas Rees was the  first member to have its crystal structure determined and is the E. coli protein BtuCD. The glycoprotein  tapasin mediates the interaction between another member of this family and MHC I molecules; that  member is TAP. The B1, C1, and G2 members of this protein family are responsible for the vast majority of  multidrug resistant tumor cel s. Important motifs in this family include the P-loop, Walker B motif, Q-loop,  H-motif, and their namesake signature motif. Mutations in another member of this family that transfers  fatty acids to peroxisomes causes adrenoleukodystrophy. A mutation in CFTR, yet another member of  this family, causes cystic fibrosis. For 10 points, identify this largest family of transmembrane proteins  that use the energy from binding and hydrolyzing ATP to transport molecules.
A member of this protein family crystal ized by Kaspar Locher, Al en Lee, and Douglas Rees was the  first member to have its crystal structure determined and is the E. coli protein BtuCD.