It blocks further stimulation of Th1differentiation by DC1 cells, and it is important in regulating the Th2 response. Its production is stimulated by interleukin-12, and only one type exists in humans. Released by a type of helper-T cell, it induces inflammation by recruiting leukocytes to the site of infection, as well as 
It blocks further stimulation of Th1differentiation by DC1 cells, and it is important in regulating the Th2 response. Its production is stimulated by interleukin-12, and only one type exists in humans. Released by a type of helper-T cell, it induces inflammation by recruiting leukocytes to the site of infection, as well as stimulating macrophages to kill engulfed bacteria. Once used as a treatment for multiple sclerosis, it was later found to exacerbate the disease. For ten points, name this disease-fighting protein that has not proven as useful in cancer treatment as interferon-alpha or interferon-beta.
It blocks further stimulation of Th1differentiation by DC1 cells, and it is important in regulating the Th2 response.
interferon-gamma or gamma interferon (prompt on “interferon” before the end)