This process is inhibited in culture by 3-methyladenine, and one method of activating this process occurs when galectin-8 binds to NDP52. ULK complexes are responsible for triggering this process, and it is inhibited by the action of Beclin-1 and PI3-Kinase. This process can be assayed for by immunoblotting for the conversion of LC3-I to LC3-II. It's not gluconeogenesis but during starvation, AMP kinase triggers this process, and mTOR inhibits this process when nutrients are plentiful. In yeast, this process is regulate
This process is inhibited in culture by 3-methyladenine, and one method of activating this process occurs when galectin-8 binds to NDP52. ULK complexes are responsible for triggering this process, and it is inhibited by the action of Beclin-1 and PI3-Kinase. This process can be assayed for by immunoblotting for the conversion of LC3-I to LC3-II. It's not gluconeogenesis but during starvation, AMP kinase triggers this process, and mTOR inhibits this process when nutrients are plentiful. In yeast, this process is regulated by the (*) ATG proteins. This process is responsible for clearing out mutant htt in patients with Huntington's disease. It results in the creation of a double-membraned vesicle around the targeted organelle, which then fuses with a lysosome. For 10 points, name this process in which a cell eats its own damaged components.
This process is inhibited in culture by 3-methyladenine, and one method of activating this process occurs when galectin-8 binds to NDP52.
autophagy [accept autophagocytosis]