Serine proteases, such as urokinase-type plasminogen activator, and matrix metalloproteinases help them form, as they clear paths into which these structures can grow.
Serine proteases, such as urokinase-type plasminogen activator, and matrix metalloproteinases help them form, as they clear paths into which these structures can grow. Their patterns can be determined by the "stickiness" of endothelial cells, (*) as some organs are more prone to have them form thanks to different cell adhesion molecules. The protein "Twist" has recently been found to play a large role in their formation, as it turns off the gene for the protein E-cadherin, and thus allows malignant cells to "slip away." FTP, name these secondary growths formed from a primary tumor somewhere else in the body, the main form in which cancer spreads.
Serine proteases, such as urokinase-type plasminogen activator, and matrix metalloproteinases help them form, as they clear paths into which these structures can grow.
{metastases} (or {metastasis}) (prompt on {tumors} or {cancer})