Stimulation of this neurotransmitter's 2A receptors is thought to explain the hallucinogenic effects of LSD, and its release is stimulated by the appetite suppressant fenfluramine.
Stimulation of this neurotransmitter's 2A receptors is thought to explain the hallucinogenic effects of LSD, and its release is stimulated by the appetite suppressant fenfluramine. The enzyme transglutinase can add this neurotransmitter to proteins, effects of which include reducing the exocytosis of thrombocytes. In the body, it is produced by and stored in enterochromaffin cells while most neurons releasing this indolamine in the central nervous system originate in the raphé nuclei. This neurotransmitter breaks down to yield the metabolite 5-HIAA in the presence of (*) MAO and its transporters are targeted by TCAs, causing symptoms like fever, tachycardia and confusion as seen in its namesake syndrome, or "storm". Commonly referred to as 5-HT, or 5-hydroxytryptamine, it is synthesized from tryptophan. Setraline and escitalopram potentiate the effects of, FTP, what neurotransmitter whose reuptake is blocked by SSRIs?
Stimulation of this neurotransmitter's 2A receptors is thought to explain the hallucinogenic effects of LSD, and its release is stimulated by the appetite suppressant fenfluramine.
{serotonin} (accept {5-HT} or {5-hydroxytryptamine} before they are mentioned)