A genetic defect in their synthesis is implicated in Zellweger syndrome, where the body is unable to oxidize certain long-chain fatty acids. Though proteins are not thought to use the endoplasmic reticulum to reach it, new evidence suggests it may have evolved from proteins related to that organelle, casting doubts on its supposed endosymbiontic origin. Key to production of the myelin sheath, it also plays a role in bile 
A genetic defect in their synthesis is implicated in Zellweger syndrome, where the body is unable to oxidize certain long-chain fatty acids. Though proteins are not thought to use the endoplasmic reticulum to reach it, new evidence suggests it may have evolved from proteins related to that organelle, casting doubts on its supposed endosymbiontic origin. Key to production of the myelin sheath, it also plays a role in bile production as well as primarily breaking down ethanol into acetaldehyde and using enzymes such a catalase to deal with toxic waste products. For 10 points, name this cellular organelle, a major produce of hydrogen peroxide.
A genetic defect in their synthesis is implicated in Zellweger syndrome, where the body is unable to oxidize certain long-chain fatty acids.
{peroxisomes}