IgLONs are a rare dimeric protein that is attached to this molecule, a variant of which is the low-molecular weight antigen that induces IgM responses against Toxoplasma gondii. Before transport, this molecule is deacylated by PGAP1. Hyperphosphatemia-mental retardation syndrome is caused by a mutation in the biosynthetic pathway that creates it. Decay-accelerating factor and CD59 attach to this molecule, which is why absence of it causes paroxysmal nocturnal hemoglobinuria. It attaches to the omega site of particular proteins, including alkaline phosphatase. This molecule, which contains ethanolamine-phosphate and mannose, helps localize substrates to lipid rafts an
IgLONs are a rare dimeric protein that is attached to this molecule, a variant of which is the low-molecular weight antigen that induces IgM responses against Toxoplasma gondii. Before transport, this molecule is deacylated by PGAP1. Hyperphosphatemia-mental retardation syndrome is caused by a mutation in the biosynthetic pathway that creates it. Decay-accelerating factor and CD59 attach to this molecule, which is why absence of it causes paroxysmal nocturnal hemoglobinuria. It attaches to the omega site of particular proteins, including alkaline phosphatase. This molecule, which contains ethanolamine-phosphate and mannose, helps localize substrates to lipid rafts and is cleaved by phospholipase C. Acetylcholinesterase and PrPc are among the proteins that attach to this molecule, which is attached by a transamidase to the C-terminus of proteins in the ER. For 10 points, name this glycolipid anchor that tethers proteins to the extracellular side of a cell membrane, which as its name suggests contains sugar groups, phosphate, and inositol.
IgLONs are a rare dimeric protein that is attached to this molecule, a variant of which is the low-molecular weight antigen that induces IgM responses against Toxoplasma gondii.
GPI or glycophosphatidylinositol