Potentiators of this process include rapamycin, which induces formation of complexes of Atg proteins critical for induction of it.
Techniques used to study this process involve detecting the breakdown of the LC3 protein, localization of LC3 to eponymous bodies, and direct identification of these bodies by their characteristic double-membrane form. Potentiators of this process include rapamycin, which induces formation of complexes of Atg proteins critical for induction of it. Too much of it can cause type II programmed cell death, although in cancers it can also promote cellular survival. Primarily induced in response to low nutrient levels, FTP, name this cellular process in which old organelles are surrounded by a membrane and fused to lysosomes for recycling, which takes its name from the Latin for "self-eating."
Techniques used to study this process involve detecting the breakdown of the LC3 protein, localization of LC3 to eponymous bodies, and direct identification of these bodies by their characteristic double-membrane form.
autophagy (accept type II programmed cell death before it is mentioned)