Although its exact mechanism is unknown, the physiopathology suggests that intraluminal lesions alter normal peristalsis of the compromised segment and acts as the head of the intussuceptum.
Aunque el mecanismo exacto es desconocido, la fisiopatología sugerida es que lesiones intraluminales alteran la peristalsis normal del segmento comprometido y actúan como cabeza del intususceptum.