The pain of prebiotics?

Here’s a question I hear on occasion: “I started the prebiotic fibers but I experienced a lot of bloating and abdominal discomfort and had to stop. Maybe they’re not for me.” As the Wheat Belly conversation is taken further into the mainstream population via the Wheat Belly 10-Day Grain Detox, I am hearing this more and more. Giving up too soon because of encountering a problem without understanding why is a sure way to booby-trap your return to health. Should you encounter problems such as bloating or discomfort with your prebiotic regimen, it is due to dysbiosis, disrupted bowel flora from prior eating habits and other factors. In other words, your previous diet of wheat, grains, and sugars, along with chlorinated/fluoridated drinking water, prescription antibiotics, herbicides and pesticides in food, etc. changed the composition of bowel flora, perhaps even allowing unhealthy species to ascend up the small intestine, occasionally as high as the duodenum and stomach (“small intestinal bacterial overgrowth,” or SIBO). Undesirable species such as E. coli, Enterobacter, and various Clostridia species proliferate and muscle out desirable species such as Lactobacillus. This can happen with green bananas, raw white potatoes, inulin, FOS, galactooligosacchrides, or any other form of prebiotic fiber. Dysbiosis at the start of your program is common. Even by lax criteria, 35% of people without abdominal symptoms can start with dysbiosis. Compared ...
Source: Wheat Belly Blog - Category: Cardiology Authors: Tags: Wheat Belly Lifestyle bowel flora cholesterol dysbiosis health hypertension Inflammation prebiotic fibers resistant starch Weight Loss Source Type: blogs

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Authors: Park HJ, Shin KC, Yoou SK, Kang M, Gon Kim J, Jun Sung D, Yu W, Lee Y, Hea Kim S, Min Bae Y, Park SW Abstract Oxidative stress is associated with many cardiovascular diseases, such as hypertension and arteriosclerosis. Oxidative stress reportedly activates the L-type voltage-gated calcium channel (VDCCL) and elevates [Ca2+]i in many cells. However, how oxidative stress activates VDCCL under clinical setting and the consequence for arteries are unclear. Here, we examined the hypothesis that hydrogen peroxide (H2O2) regulates membrane potential (Em) by altering Na+ influx through cation channels, which conse...
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