Early Symptoms of Lou Gehrig Disease

early symptoms of lou gehrig disease
What are the early symptoms of Lou Gehrig disease?

ALS is a motor neuron disease neurodegenerative nerve cells in the brain and spinal cord that regulate voluntary muscle movement. ALS also known as Lou Gehrig’s disease. In ALS, nerve cells (neurons) throw-away or die, and can no longer send messages to muscles. Early symptoms of Lou Gehrig disease eventually causes muscle weakening, twitch, and the inability to move arms, legs, and body.

The condition is slowly getting worse. When the tissues in the chest area stop working, it becomes difficult or impossible to breathe normally. Though there is no recognized cure, there are some natural supplements that can take to slow the progression of early symptoms of Lou Gehrig disease.

As far as possible causes of ALS, there have been studies linking aluminum content water supply for Lou Gehrig’s disease. Aluminum can interfere with the absorption of phosphate cause hypophosphatemia. Also, apoptosis is a different form of cell death controlled by an internal program coded suicide. Apoptosis also accounts for many of the pathology seen in Lou Gehrig’s disease. Also, characterized by loss of neurons of the brain.

In specific, the part of the Axon nerve damage, leading to a proximal axonopathy. Axonopathies proximal often caused by volatile organic compounds. Which reduces the Involuntary neurons with stabilizing membrane and decrease the ion flux. The altitude in the death cell or apoptosis appears to be related to the lack of production of nerve growth factor and damage of free radicals.

How to treat early symptoms of Lou Gehrig disease?

The manipulation of the process of cell killing and supports the function of the immune system of a healthy body helps in treating early symptoms of Lou Gehrig disease. Long-term nerve growth factor delivery will protect programmed cell death in individuals who suffer from Lou Gehrig’s disease.

An individual with the dominant form of familial amyotrophic lateral sclerosis has reduced gene defects cytoplasmic superoxide dismutase (SOD) by 40%. The shape of the cytoplasm SOD requires copper and zinc. Evidence for the role of SOD in preventing free radical damage from different sources. Unlike the mammalian enzyme, coated, non-soy, legume-derived pancreatic enzyme action rejects the SOD, and the study double-blind. Also, placebo-controlled study suggested that oral consumption of this supplement can increase the activity of SOD in a time of 4 hours.

There is a strong correlation between heavy metal toxicity and neurological diseases. If you suspect heavy metal toxicity, a detoxification program is too cumbersome metal poisoning.

Fatty acids required for health and that cannot be done by the Agency are referred to as essential fatty acids (EFA). EFA aid in the transmission of nerve impulses and is necessary for healthy development and functioning of the brain. There are some roots of essential fatty acids; such as fish oils, flax seed oil, grape seed oil and evening primrose oil.

In short, if someone you love, has given a diagnosis of early symptoms of Lou Gehrig disease. You may benefit from supplementation with S.O.D., copper and zinc, essential fatty acids. Also, as well as a live heavy metal detoxification program.