You apply first aid to yourself It's got a what's stopping the bleeding the pressure band.


What's up Chicago quick experiment that I like to do especially because I started talking to a few people and Heard some interesting company name. So just out of my own curiosity. How many people by show of hands are in a company with a hundred or more employees? Okay, keep your hands up 500 more employees a thousand or more employees Five thousand or more employees Wow, ten thousand or more employees All right, cool. All right, put your hands down My name is Jeff My talk today is hacking human systems and this is a different talk for me because typically when I give a talk I'm talking about something that I've done or I've implemented or some strategy that I've been working on this is largely going to be like a 400 person stream of consciousness as I kind of Dissect this idea that I've been working on about like how human systems work how they're cornered sort of at the center of everything that we Do I think people in the larger organizations will probably take some of this away big time just because of the whole Let's say bureaucracy of large organizations I'm gonna say a lot of things that you might sit back and be like, you know what I want to fight that dude All right, I'm gonna fight that dude because I don't believe what he's saying That's fine. Like I said, this is a thought experiment. So please, you know catch me outside and We can chat about it either, you know during the open spaces or after the event But please I want to start a dialogue about this stuff So if I see something and you're like, I'm cold and bullshit Please do it because I think starting a conversation about this stuff is really key Oh, I forgot to my $500 stopwatch. All right. Sorry. Just want to make sure we got time. So What are we gonna be doing we're gonna I'll introduce myself We'll talk about systems and what systems thinking is we'll walk through a traditional system that I think everyone should be familiar with Hopefully because you're getting paid We'll talk to examples of human systems in our field and redesigning our approach to solving problems Sorry, it's new clicker. I'm borrowing it from someone. So my name is Jeff Smith I work for a company called Centro that you've never heard of that you both love and hate I'll tell you why I'm the manager of production operations there. I've got a small team of about three or four engineers And we are supporting a Ruby on rails application prior to that I was with Grubhub for a number of years helping them build out their SRE team. You can find me on Twitter. Where we at? All right, you can find me on Twitter at dark and nerdy Feel free to email me. I also have a blog that I don't update all things dark calm I write a lot of medium posts to there so you can get links there if you want to reach out to me But I'm not hiding. So there's plenty of ways to find me Centro is a leading developer of digital advertising software. That's why I say you both love them and hate them You hate them because we help make digital advertisers more effective You love them because you don't pay a monthly subscription to Google search So that's pretty much how I sleep at night advertising is pretty much what powers the free internet. So That's what Centro does So Docker What does Docker have to do with systems thinking absolutely nothing? I just wanted to get your attention I notice everyone lean forward Right, so it's officially a DevOps talk. We can move on but we said it right? I can now officially label this appropriately and slide share So what is the system? Systems are sort of all around us and when you start talking about systems thinking you start to see How the world is made up and connected with all of these different systems and in the technology field We tend to focus on computer systems networking systems But the interesting thing is most of you don't have technology problems Most of your problems stem from the human systems that are throughout your organization, right? No one is like we don't know how to implement a cache layer That doesn't happen. It's not about the technology It's about getting the momentum to actually get the organization to move to do the thing that you wanted to do government systems Prioritization Resource management these are all human systems that are actually impacting your ability to deliver on your technological goals But for some reason we sort of ignore them and we don't pay attention to them And we don't really know how to navigate And manipulate them the people that do are called, you know playing office politics But in reality those people that are playing office politics are getting shit done a lot of times Or the very least gonna raise So what is the definition of a system a system is an interconnected set of elements that is coherently organized in a way that achieves something And we're gonna focus on the something part because that part can be a little Misleading or disingenuous when we think about systems. So there's three components of a system You've got elements interconnections and purpose The elements of a system are all of the parts the resources the agents the people the computer systems They're all of the things in the system in which we measure the stocks Which we'll talk about a little bit that we measure to know how the system is behaving or perform Interconnections are the connections between those elements. How do those relationships impact the way the system performs and then there's purpose What is the point of the system?  Think about since we're in Chicago think about the beautiful soda beverage tax, right? The stated goal is we're going to reduce calorie consumption of our constituents We're gonna make them healthier and live a more active lifestyle. No, we're paying for pensions That's the point of the system and when you look at it from that perspective You'll notice that all the elements and the interconnections of that system are focused and targeted to that goal So it's very important that when we look at these systems we think about what is it that it's actually doing versus what it is That we claim it's doing So at the heart of all systems are stocks and Stocks are the elements of the system that you can feel count or measure at any given time We're gonna put a lot of emphasis on feel Because so much of what happens in some of these human systems centers around emotions Emotions of all types typically emotion is at the center of something We like to pretend that we are Logical decision-makers, but in reality it's emotion. That's at the center of the decision and then we use logic to back that up So here's the basic stocks how a stock basically works in a system, right? We'll take our bank account. I Do have more than a hundred dollars in my bank account. I was just No, I don't We've got the stock our bank account our dollar amount And we have inflows and outflows to that stock inflows deposit increase the stock withdrawals Decrease the stock and the system is largely calibrated Based on the number based on the value of that stock We have this thing called feedback loops and a feedback loop is formed when changes in the stock Effect the flow into or out of that same stock, right? So continuing with that same example, we have a concept of a balancing loop as Your bank account gets lower $25 is probably more accurate for me You work more hours so that you can increase the number of deposits You might also slow down your withdrawals Right once your bank account starts getting low. You're like, all right. Maybe we won't go to Ocevall this week We'll go to Burger King or McDonald's That's called a balancing loop it make sure that that stock stays at a at a reasonable level Balancing loops to work the other way too. Now if you got a million dollars in your bank account, what could happen? You might work less hours You might spend more but you're probably going to spend more right no point in letting that cash sit in there Might as well get a boat Something you can do Then we have this concept of what we call a reinforcing feedback loop and Reinforcing feedback loops are when the stock is able to generate more of itself simply through its existing Common example of that is your interest rate or your the interest rate on a savings account right as the stock grows Your interest rate is a percentage of that and you get more stock Sticking with the Chicago examples corruption Corruption is a reinforcing feedback loop the more corruption you have the more it generates of itself and on and on and on and on So we're going to talk about everyone's favorite system the change control system How many have some sort of change control process in their organization? Okay, not as many as I would have thought Get on that shit guys the interesting thing about a change control system and and I'm being very Broad stroke and generic here, but most change control systems that I've seen Don't actually implement the stated goal of what they're trying to do right they are They are subject to a bunch of other factors that make the system behave in a different way Now the stated goal of a change control system is to introduce Changes to the system in a controlled fashion with mineral minimal disruptions. We talk a lot about risk management, right? That's always a big thing But when we talk about Change management, and we look at the actors and the agents the elements in the system We realize that sometimes the participants in the system aren't the people participants we need to deliver the stated goal or outcome so here's just like a Rough sketch I did of a generic change control system, right? We've got a bunch of business changes business operations that happen. So our two stocks in the system are changes in risk Business operations generate changes When we have enough changes or through some regularly scheduled process We have this change approval process right this cab board the cab board approves or denies those changes and Those change approvals or denials increase risk Once risk gets to a certain point. We do some due diligence. What does that due diligence usually look like? It usually looks like a manager who is way too high to actually know what's going on about a thing Going and talking to the SME that just submitted the change, right? Do you think this is dangerous? Yes That's why I submitted it It's gonna take the whole system down trust me don't do what I just asked you to do it's crazy So we end up wasting a bunch of time so that the person that originally asked to do a thing is asked if they should do a thing So it makes you wonder like What is this piece actually doing? It's risk management theater in a lot of ways And if it's risk management theater if we're not actually reducing the risk Then what is the actual point of the system? in my worldview This system is typically about limiting the amount and frequency of change introduced to the environment That's what we're actually talking about Because what happens when the change goes bad, right when that networking change goes bad, it's like all right guys We're only going to do networking changes on Thursday. Okay, because that's reducing the risk, right like sure We're not taking it down during business hours, right, but they're still an outage Then it moves to all right. We're only doing it on Thursdays at 2 a.m And it's like great now. I'm tired and I'm doing this networking change That's really reducing risk My favorite is the security audit so that the inspiration for this talk started at DevOps day Seattle We were in an open space and we were talking about Security processes and how we manage a security controls and how frustrating that audit processes And we're all sitting there griping about the different types of others We had and these things that they were asking us to do and how we had to document it and I stopped and I thought I said well None of us have actually challenged the validity of these things that they're asking for And we quickly all agreed like well, yes shit. We should be doing So then why are we so up and angst about this ask? Why is it that we wait until the audit comes to be able to like spur into action And do all of these things to make sure our audit assets are covered So and thinking about it. I kind of give it this term audit anxiety, right? There's usually a bunch of things that you have to do in an audit, right? Make sure you got your documented policies. You got to make sure you're enforcing those policies You got to have proof that you're enforcing those policies show me when this thing happened and how you handled it And then also you have to prioritize these fixes But why is it that we wait for the audit to actually start to do these things in earnest? And typically the larger your organization is the slower this rate of change unless you have like a Security czar that is like always down your throat about it I got a guy that a friend of mine. He's got a five-person team and three of them are dedicated to security, right? So that shows you the the sort of Prioritization that if nothing else the boss puts on the organization So I started thinking about it and I tried to come up with like a system flow of what that might look like So our stock is sort of our known risks, right? That's the thing that we're measuring and Through different activities. We create prioritization pressure which flows into those known risks, okay? So internal findings is a big one, right?  You notice that, you know, I don't know Open SSH hasn't been patched recently or something to that effect That creates pending security work that now has a backlog that now is documented somewhere So we're like, okay, we know about all these risks. You also have publicized exploits, right? Something that's out there in the wild people hear about it. Suddenly. It's a big deal But then you also have the time until your next audit, right? And the more the less time there is to your next audit the more prioritization pressure Builds up why because you now have identified these known risks And we decrease the outflow for that stock is the actual security work actually implementing these fixes and changes But the interesting thing is that prioritization pressure isn't necessarily Logically Equivalent to the actual finding And there was no better example of that than Heartbleed, right? When Heartbleed came out It wasn't the worst security vulnerability we've ever seen but you know what it did have a kick-ass logo Right it had a kick-ass logo and that kick-ass logo got it on the news and once it got on the news everybody's boss was like Hey, you hear about this Heartbleed thing. You're like Yeah, but we've got like 15 other vulnerabilities I've been telling you about for six years that we need to patch how many people patched Heartbleed within the first week Is that a normal rate for you? Heartbleed had a had a Mismatched amount of prioritization pressure, but it moved the organization in a way that forced you into action So I started thinking about that and I started thinking like how can we? Manipulate our systems to produce that on a regular basis We can mimic other traits of we can mimic the traits of other elements How do we sort of create that buzz inside of our organization that we created with Heartbleed? Might be hard because we don't have newscasters on our side But maybe we could get Tom skilling or someone to just drop a note in the middle of his weather forecast like oh, and there's a new vulnerability for Apache We can alter the interconnections between elements in the system What if we change the reporting structure in terms of who's notified when a vulnerability is found Or what if we could change who's on the hook for when a vulnerability is found and not patched? What if we change the rules all together? What if we said as An organization we're going to commit to publicly disclosing any vulnerabilities that we have that we haven't addressed within 60 days ourselves That generates a lot of pressure internally right because now you're saying all right We're going to air our dirty laundry if we don't fix our shit now I don't know how you're going to find a CTO that's going to agree to this but It's an interesting thought experiment to figure out like okay What are the levers we can kind of pull to force or push our system into action? Docker just make sure you're with me make sure you're still paying attention right. We're still not talking about it, but Feel free to lean forward Configuration management my favorite how many people are using some sort of configuration management salt puppet chef Ansible okay, not as many as I would hope how many people are working on it right now go ahead raise your hands up Come on okay, and how many people are finding that it's not technologically problematic, but more Resource planning or prioritization to actually get that implementation going Because config management is a pretty easy sell right So the fact that we're struggling to actually get it implemented everywhere is sort of like a tell-tale sign of exactly what I'm talking about So what is awesome about config management all this common stuff that we hear it's easy to recreate environments We get traceability of changes We get source code management that allows us to help say you know who implemented this change to the discussion around it peer review The repeatability aspect is awesome because you know even though there's a command that we're supposed to execute I guarantee you if you've got ten servers Bob is going to screw up server eight and You're going to be wondering what the hell is going wrong with one eighth of all of our requests So the benefits of config management are pretty easy. So what is it that drives us this sort of adoption a lot of times? our stock in that system is the pain of failure and What drives the pain of failure well sort of your rebuild frequency right? Company growth is another one as the company grows you've got to add more servers that becomes more painful than you say man Why are we doing this when there's 15 tools out there that will help us with this and eventually you start working your way towards automation? So again, how can we sort of simulate this thing because like hardware failure is sort of out of your control And unless you're a large organization like Google chances are you're not experiencing hardware failures that much right like the difference between like Google and You know, I don't know the uber of knitting They're gonna have very different like hardware failure rates Company growth kind of outside of your control as well. So how can we manipulate the system in a way to do that? Well, someone has already done it for us. Who's familiar with our buddy chaos monkey? Right chaos monkey is gonna go around and kill servers at random The whole point of it is to make sure that you can rebuild these servers quickly and that your systems can handle the failure With chaos monkey, you have knobs that you control how frequently you're going to do it So you can now manipulate the inflow into the pain of failure And you can say look boss like you know, these are the things that are happening in our environment And this is problematic now I'm not saying it's as easy as you know firing up a VM launching chaos monkey and walking away That'll get you fired, but You know, it's another example of like how we can take the system Manipulate our inflows to try to produce expected behaviors And that's really what we're talking about We're talking about manipulating the system to provide our desired behaviors and a lot of time that is gonna be a play on emotions Whenever we make a business case for something they always say, you know, oh look at the ROI build on the ROI and push that out No, that doesn't always sell it right what sells it is the fire and brinstone, right? What sells it is the boogeyman tails, right? Well, you know, if we don't patch this some 400 pound hacker is gonna be able to manipulate our systems and take everything down suddenly now We're ready to spring to action doesn't matter how true or false that might be We're generating the emotion of fear of panic and then we're backing that up with logical reason So how do we look at these systems and sort of artificially create that behavior? So I wrote a post called I don't understand immutable infrastructure because I don't really understand immutable infrastructure And what I mean by that is I don't necessarily subscribe to all of the reasons why we say we don't want to change a running system I can think of a number of reasons why I want to quickly manipulate a running system so I put it on Twitter like you do and Naturally someone wanted to fight me so we had a nice hundred and forty character brawl and at the end of it he said something that Made everything much more clear to me and sort of is is is sort of the point of this conversation He said to oversimplify it's the difference between you can automate everything to you must automate everything Despite all of the rhetoric or or BS and remember we can fight outside later Regarding immutable infrastructure this stuck to me because it's like Okay, he's really using immutable infrastructure to force a behavior that he couldn't consistently get before He's using it to say hey guys if you don't automate this thing Right, you're not going to be able to get your change out into the world Because you can't log in you can't SSH into this server and just make a change you just can't do it You have to automate it and that really stuck to me because that's it All right, this is exactly what we're doing. We're again changing the system to behave a specific way so that we can elicit behavior Because even when you have config management, it is always especially especially in times of crisis It's always easy to say I'm gonna make this change real quick and then I'll Backport the change in the config management, right? I'll do it hot and then I'll do a fast follow Right, and then someone says hey, man.  Yeah All right So now your elbows deep and some barbecue and you're like did I do that?   We got to fix this start closing tickets and there's all right canceled canceled not gonna fix not gonna fix and Somehow that brings about a sense of calm, right?  We didn't do Thank goodness, but that's not helping anybody.  I got ahead of myself I actually said what I was supposed to say in this slide in the previous slide So I'm just gonna spend a minute looking at you and then we're gonna move on We make decisions on emotion tapping the emotion drive the behavior So what do we talk about? Systems are made of elements interconnections and purpose Those interconnections those elements are things that we can manipulate in order to create our expected behaviors Stocks are the basis of any system and it's often the most difficult part to identify in a human system What are these stocks that we're playing around with? feedback loops balancing loops and reinforcing loops balancing loops help keep our stock at a reasonable level we get the deal what reasonable is reinforcing loops are things that Allow the stock to grow on its own corruption All right tech debt Tech debt breeds more tech debt right because you're building poop on top of poop Make poop tackle problems by focusing on what feeds or drains our stocks Understand the inflows and outflows of those emotions Because those are key to managing our human systems. Thank you. Hey, thanks chef That was a great talk. My name is Jesse Martin from Eli Lilly and company in Indianapolis And actually I had more of a comment actually because I thought your comment around how do you take some of this security patching? seriously something that we actually had to do was we have Kind of this culture of you've got to incorporate security in your application development supports problem or quality system We actually had to create a separate kind of infosec organizations with deep experts that were really paying attention to this because exactly to your point you just couldn't get Focus on these things without an organization and kind of people that were attending the you know the Various hacker conferences and really really current on security to actually raise the alarm and actually have very very senior kind of executive Reporting responsibilities that is actually separate from your quality organization to just kind of lend a hand So that's something that we haven't I'd say it's kind of it's working for us so far, right? Yeah That's a common pattern that you're starting to see Specifically, you know doing it in sort of like a DevOps structure where you know these infosec people as opposed to being these outlets that are on top Screaming about what you do their partners and they go in and they help you because in reality the developer is not incentivized through security He's incentivized through features through shipping code and when those things aren't in alignment Which is a common problem in all organizations is incentive alignment when those things aren't aligned That's exactly what you ran into so having a separate work to partner with those guys and to say like hey Let me help make it easy for you Yeah, that's always a winning strategy So I'm just curious how you can I Any tips on identifying what the actual stocks in the system are Yeah, so when I was actually writing this presentation and thinking about like change control and things like that really it was about thinking of the times when Something move the system in a direction that it was uncommon, right? You sort of have the system that sort of stagnant You say yeah, I know change control is always going to take me a week and a half because that's just the process But then look at the items that are the outliers and say okay So what was different about this situation that made the system behave in a particular way? And when you think about that and try to break up all of the parts that were involved in that decision usually The stock sort of like jumps out at you and you says oh it was because there was all of this fear around heart bleed Potentially opening up our world to a world of hurt, and it wasn't necessarily that it was actually More dangerous than any of other of our other vulnerabilities It was packaged better and whenever you talk about packaging a problem and selling it You're talking about some emotion of either fear or joy And then you just tap into that It's not a science by any means great. Thank you so much Jeff. That was awesome


Commuters plying the Asaba-Ugedi Expressway were stranded for several hours and this was after a protest by Uvrode community in Isoku North local government area over alleged neglect of the community. The protesters besieged the expressway for hours. They looted placards with inscriptions such as Uvrode is bleeding and no federal and state government projects in Uvrode community. There are inscriptions where our grammar school is in bad shape. No teachers and no chairs. No constituency projects in Uvrode community. Oil exploration in our community since 1963 till date and nothing to show for it. President-General of Uvrode community is Mr. Felix Efedoma. He said both states and federal governments have abandoned Uvrode community despite being an oil producing community since 1963 till date.


It looks like we're live, everybody. You are in for such a special treat today. I'm here with Dr. Corson and I'm gonna introduce her in just a moment here. She's a wonderful guest and we haven't known each other that long although we've been in the same circles for years. So I've known of her and have had the greatest respect for her work and the lecture she's just given recently about Fibrin. We're gonna dive into that in just a minute or two. But before we do, I wanna give you just a little background. You all know me, you've been here before.  And there's like 60 plus hours of interviews with great experts like Dr. Corson here. And that's just on my YouTube channel under my name, Jill Carnahan. If you do go there and enjoy them, please subscribe so you stay updated on the new content. And this video, depending on where you're watching it, will either land on YouTube in a week or so or maybe you're watching live. Other background, if you want other information on blogs or things on my website, it's just jillcarnahan.com. And I don't often mention products, but if we do, you can find those on drjilhealth.com. So I am absolutely delighted to introduce Dr. Ann Folt Corson. She grew up in Southeastern Pennsylvania and obtained the Doctor of Medicine degree in 1982 from the University of Pennsylvania School of Medicine in Philadelphia. She's been board certified in family medicine, just like me. Since 1983 and Integrative Holistic Medicine. So we have the same credentialing too. Integrative Family Medicine is a unique field that combines modern Western medical science, traditional herbal medicines and nutrition as informed by philosophies of ancient Chinese medicine. And again, we practice very similar too. What I'm sure with Dr. Corson and I both have seen is we have this wonderful foundation of Western science but then it's like our toolbox is bigger now that we're able to use all these other modalities as well to help people. And I don't know about you, Dr. Corson, but I find we need those tools, right? We need everything possible nowadays with a complex chronic illness. She approaches environmental, infectious, dietary, metabolic, biochemical, endocrine, immunological, structural, emotional and spiritual detriments to disease. And her practice focuses on identifying and addressing the root cause of each patient's illness. She has a solo practice in Philadelphia and we were both just saying before we got on here how gosh, the patients are coming out of the woodwork. It's actually really hard to keep up. I think that as COVID had people packed away, they're starting to come back out and realize how important health is. We're both just realizing the need to educate.  She has so many awards, credentials, she's been speaking around the world. I won't read the rest of this here but you can find her and your website, Dr. Corson, where can they find you? AnnaCorsonandd.com, it's a very rudimentary website. So don't expect much from it at all. That's okay. Well, we are so delighted to have you here. So welcome and thanks for coming. And before we jump into the fuss about fiber and what this is all about, why it's so important to the people listening to if you're a practitioner or a patient. I would love to hear just a little bit about your journey is very, very parallel to mine with the family medicine and then the holistic integrative. And how did you get into medicine and how did you get into integrative medicine? Tell us a little bit about your story. Well, I come from a long line of doctors and for some reason it was my siblings, it was always me that was gonna be the doctor. Although at first I thought I was gonna be a neuroanatomist and so I sort of rebelled and went to graduate school neuroanatomy before turning around to go to medical school. But I just seemed to fall into that naturally. And the nervous system was my first love. So I was thinking neurology or something like that and I started out that way. But then I really found that that was very limiting. So I actually became an emergency physician and I practiced emergency medicine for close to 15 years, not quite. And then my only child got quite ill and it took me a couple of years to figure out what it was. And I remember complaining to a friend of mine one day, I can't figure out what's wrong with him. And he said, well, he's got Lyme disease. Look at the symptoms. I said, well, I've done the Eliza three times and it's come out negative, right? That's what the American Academy of Family Medicine really tells us to do. And so he gave me a flyer for a lecture that Dr. Joe Bristano was giving on the 3rd of May or the 8th of May. I can't remember quite, 2003 at the local high school here in Chester County, Pennsylvania. I went and I was just so enthralled. I learned more in that lecture than I had in years of going to medical meetings and whatnot. And so I realized that my son had Lyme disease in 2003. So I immediately, within just a little short time after that lecture asked Bristano if I could come visit him in his office because that's the way we learned things in medicine, he wanted to do one teach one, right? So he had to learn. And so he said, well, that's okay. No one's ever done that. I started the whole physician training program with ILADS. Wow. One to ever ask. And he said, you know, next week I've got somebody coming from Wales. And so David Owens came the next week. So I spent time learning with him. I had gone back, I went first in July and then went back in September of 03. Then a couple of years later, I went and trained with Charles Ray Jones in Connecticut. But man, I just hit the floor running in the summer of 2003 with Lyme disease and just started my own treatment. I've been an ER doctor and I've done primary care in family practice offices and work in its comp situation. I've helped in the OR and I worked ER. So I had a lot of primary care clinical experience. And so, but I found that these were the most complicated patients you could imagine. And then in 2004, I was a local psychiatrist by the name of James Shaller that was sharing patients with me. And he said, you also have a mold problem, Ann. I said, what do you mean? So he gave me a pre-print copy of Mold Warriors. So I learned about mold in 2004. So I started treating mold in 2004. And then I had a patient come to me and she put a bottle of Toxics on my desk, which is a fecana product. And she says, I don't know what this stuff is, but this is really good. S blank, blank, blank. She says, I took two drops of it and I was sick for a day and a half. So I said, look, I looked this up. And so I looked up the company and I started exploring all of these natural remedies. And I learned about German biological medicine and I also just jumped right in the deep end. And I went to all these trainings by Gary and Rain Klepper and some of these other people and got into sort of that Northern California naturopath community. And I just found my tribe. And so that was in 2005. So I learned about hypercardiability way, way back in 2005 from Gary Klepper, who learned it from David Berg a couple of years prior. So I just, again, I just hit the floor running and kept going. And I've just found a wonderful armamentarium of medicines and things to use for my patients. And then just have been growing and growing and growing all these years. And it's just been a blast because it's so much fun to help whole families get better, you know. Oh, what a beautiful journey. And it's so driven by, same as my journey by this inquiring and curiosity, right? When we hit, again, we've got a great training background and I have all the respect in the world for our allopathic training and medical school. And yet we ran into limitations in our own lives and it's like, what else is possible? And then what else is out there? And then we start to realize, oh, there's a whole nother world of ability to treat and help. And I feel like it's just the best way to really combine Western allopathic medicine with all of these other things because they really add, like you, I learned so much from my naturopathic friends. So hypercoagulability, let's first define it. But I just wanna say my personal experience, I've known about this, I've known how it correlates with Lyme and complex chronic illness like mold and COVID. And so I want you to describe a little bit about what does that mean for people who are lay persons, vibrant and hypercoagulability. But I will say, I felt like this is one of those really missing links in a lot of practitioners toolboxes to really understand how much this affects so many of their chronic patients. So, yeah, go ahead. It's so true because the issue of hypercoagulability is really often overlooked even by the most experienced integrative practitioners. People will often come to me after being to many people that I respect greatly and it's one of the things that's not been looked at at all. But unfortunately it's really present to some degree or another in almost every chronic legal patient and is present in acutely ill patients as well. So think about it, everyone's had the flu, right? Everyone's had a fever, a headache, body aches and pains, fatigue, nausea, insomnia, right? A lot of those symptoms can be exacerbated by the hypercoagulability that happens when there's any inflammation in the body, right? So an acute inflammatory response from a flu can also drive, it also drives the hypercoagulable fibrin producing response in the body. So whenever we acquire it, we have a brisk innate immune system response. Then part of that innate immune system response activates the coagulation cascade, which is this domino effect where the body creates more soluble fiber. And so a lot of these symptoms can be attributed to that. And acute or chronic innate immune system activation, heavy metal toxins, mold toxins, chronic mass cell activation, percchimer reactions, inflammation of any kind can result in the generation of excess soluble fibrin. Now, so what's hypercoagulability? Well, the body maintains this wonderful balance, this seesaw balance, very delicate balance is very redundant of proteins that either encourage or discourage blood clotting, right? This is a very, very complex and redundant system and it's very, very important because after the injury to a blood vessel, we need to be able to immediately plug and stop that bleeding, but we also have to stop the formation of that clot so that we don't make so much clot that we block that blood vessel and damage tissues, right? The update, excuse me, I'm in the middle of a big thunderstorm here. So if I get interference in my internet connect, I'm on an ethernet cables, hopefully it'll be all right. So you've got to be able to either plug or thin clot or unclot all the time. Now, when this delicate seesaw is in balance due to a genetic factor, an environmental factor, a toxin infection, emotional stress, a physical stress like a physical trauma, a surgery, something like that, then it can result in too much soluble fiber being made and not being able to degrade it enough, right? So when you can't degrade it enough, you end up being sticky and that's the sort of the sticky side. And so you have an abundance of molecules that are encouraging the formation of fiber and blood clotting over blood thinning. And so these are many different causes that can precipitate us into it. Well, what is actually soluble fibering, okay? So the coagulation cascade proteins are a series of molecular reactions that result in the production of fibering. Fiber molecules can either be soluble and free-floating in the bloodstream or they can become insoluble and cross-length and that creates a clot. It takes a burst of thrombin to actually cross-length them and make them a clot. They're kind of like interlocking Lego toy pieces. Okay, that's the clot. But generally in the absence of a tear and a blood vessel wall, you don't actually get that cross-linking happening, that burst of local thrombin that causes the interlocking. So you generally just get excessive soluble fiber and produce, right, in the area. And that creates a sludgy layer on the surface of the endothelial lining of the blood vessels. And just one micron, which is one millionth of a meter, thick layer of soluble fiber along the inner lining of the capillary reduces the diffusion of oxygen molecules out of the blood into the tissues by 500% with resulting tissue hypoxia. And that was referenced in a, Nemerson had given a lecture at a 2003 meeting. I can't find the original article, but the information about Dr. Nemerson, I can give you, but Dr. David Berg references this in a lot of his lectures that he gave over the years about hyperquaggability and it's a wonderful picture of the capillary, which we can show if you'd like. But so even a small amount of soluble fibrin in the bloodstream and that sludging can create very significant clinical symptoms. And most allopathic medicine doesn't even think about it until you've got blood clot. Right. A DBT. Let's talk briefly about the symptoms, because that's where people listening, it always in that for me, that's been a big, what would a patient look like that had an issue with this? And it's probably gonna be very non-specific, right? Because there's lots of. Right, and then after that, I'd like to sort of talk about the different ways the body deals with coagulation because especially in our current era, we need to think about platelets as well as fiber formation. So the signs and symptoms of the hyperquaggability.  If you have other kind of fatigue and issues with insomnia, let me just see if I'm missing anything, hold on. Brain fog, irritability, anxiety is a big, huge one. I often will treat hyperquaggability with anxiety with fibrolytic enzymes. You can have depression, you can have mood swings, a fatigue, generalized pain that could be utterly debilitating, kind of like I just hurt everywhere. Every time you touch me, I hurt because the tissues are hypoxic. You can have painful numbness or pins and needles, sharks having shooting pains, deep aching pains, especially in the arms and legs. You have limbs that fall asleep really easily or inability to, as I say, tolerate aerobic exercise with worsening of pain and fatigue after exercise.  Often that's hyperquaggability at night. So those are sort of the major symptoms that people will have with it. Now what about the physical? What about headaches and migraines? Would you put those in that class or not necessarily? They can. That is very good to add that, especially with some of the side effects that people are now seeing after being vaccinated. The headaches and migraines can be a sign of sludging as well as serious clotting in the brain vasculature. So yes, thank you for adding that. Good. And so you talked about fibrin. You got the sticky blood. You gave symptoms and that would probably include this whole hyperquaggability, hypoxic thing.  I kind of interrupted you. Do you want to go? What I'd like to do is I'd like to talk about what the physical signs are because people can see these in themselves. Perfect. As well as the clinicians that are listening can see them in their patients. So you know how people sometimes get modeled skin when they're cold and especially as you see that in babies and infants? Well, if people have chronically modeled skin, they may be hyperquaggable. They have cold, clammy extremities but they're centrally warm. They have very prolonged capillary refill in their fingers and their toes. They can have a very pale swollen tongue with scalping of the tongue edges from indentation from the teeth. That's not just hypothyroidism. That can also be splenic congestion, hyperquaggability. And then you can have a very doughy abdomen when you examine someone's abdomen periumbilical fullness and tenderness. And you can see generalized sort of soft tissue congestion as if the tissues are just sort of body. They say, well, you know, it's like I used to be able to see my tendons in my hands and now I can't even see them anymore. Often the head and neck can be very red or ruddy in color upon laying down. And then the feet when people are sitting can be really red or purple or purple when they're sitting on the exam table and their feet are just hanging. And then there's often a very significant compromise of cognitive function, not only with irritability, but emotional liability. Excellent. Yeah, I'm sure when I feel listening with your clinician or patient or resonating, especially if you've dealt with Lyman mold, I really think, like I said, Dr. Corson, this is such an important piece of many, many of our chronic patients. And I mentioned COVID, but any virus, especially an inflammatory virus like COVID can also be in this pathway as well, correct? Yes, that's correct. It doesn't have to be, but we'll talk about that a little bit later. I don't want to get it into it right yet. Okay, so now, how does the blood coagulate? There are three main mechanisms. Number one, if your platelet behavior and whether platelets have been activated to clump, and that's separate from the protein cascades that either make fibrin or break down fibrin. So it's platelet behavior, fibrin formation and fibrin degradation. Got it. Those are the three main mechanisms that are controlling it. Now, platelets will aggregate and stick in response to trauma to the blood vessels, in response to immune system activation by toxins or infections. And one of the problems with the spike protein is that it activates platelets. Yeah, yeah. It also damages endothelial lining. That also stimulates the coagulation cascade. So you've got two things you have to think about in this scenario now, what we're currently dealing with with the spike protein, both in natural infection and vaccination, is not only platelet activation and also fibrin formation. So the treatment for that is going to be a little bit different than what we would do for just someone who's got hyperquaggability from maltoxins or heavy metal toxins or some other issue. So again, it's always this physiological balance between formation degradation. Normally platelet aggregation is pretty easy to deal with. You normally use high dose essential fatty acids, vitamin D and sometimes baby aspirin, things like that. Usually you think of platelets being pretty easy to deal with, but the intensity with platelets aggregate, given our current issues that we're dealing with in our country, we have to be very, very aggressive about that. But we have to buffer both the fibrin formation and platelet aggregation. Are you recommending anti-platelet therapy for patients who either had long-haul COVID or post-vaccination issues? Yes, but primarily very high dose vitamin essential fatty acids, I usually try to get between three to four grams a day of a very pure omega-3 and then at least 400 international units of vitamin E and depending on their symptomatology and what else they need maybe a baby aspirin a day. You really have to block platelet activation. And unfortunately, the blood test that we do to evaluate coagulation, we used to be able to get platelet activation testing, but when esoterics was bought by lab where they dropped that test. So initially David Berg had set up PMAX and then esoterics bought it and then lab core bought esoteric. So you can get these tests off your lab core, but they drop the ones that indicate whether the platelets have been activated. And that's a real problem because nowadays we really need to know what to do. Yeah, right. I've noticed that too. It's hard to get the information that we need. Yeah, it's very hard. You can get your information about your fibrin formation and degradation, your thrombin formation and degradation. And you can get some clue as to the genetic issues that people have, but it's very difficult to get information about platelets. So it's safer to treat them as if their platelets are going to be activated. Good question on that. Cause obviously with like Crohn's or colitis or some inflammatory disorders, we see thrombocytosis, which for those of you who don't know, it's excess platelet production, not necessarily how they behave would just thrombocytosis alone also contribute to issues or we have to be the activation of those excessive platelets. No, whenever you've got too many cells, of course it depends kind of how you get, you can get all kinds of sludging and blockage. Yeah. Just when you have abnormally shaped red blood cells and you've got salchemia or you have babesia and you have some other red blood cell inclusion and the red blood cells can't squeeze through the capillary beds, you're going to have some problems. Yeah. So we've talked about the symptoms of hypercrime ability. We talked about the physical signs of hypercrime ability. Again, a good way to start dealing with it is again to really help with your omega three to six ratio. Most people in this country need very high dose of omega threes for a prolonged period of time. You really need to optimize your liver detoxification pathways, but you're not going to do that until you've got dysbiosis and what I'm finding is that the vast majority of people, at least in this part of the country where we have a great deal of glyphosate used in our agricultural areas, very around all around South Eastern Pennsylvania is that their lactobacillus has been totally wiped out. It's just not growing at all in their school tests. So we need to make sure that we are fixing that gut dysbiosis by trying to restore the lactobacillus. The best way to do that is so far on finding is to really have as clean a diet as possible to bind to get rid of the glyphosate that you have in the body. You have to replace the manganese and other trace minerals because manganese is very essential for lack of the cellist to grow. And then I use a very high dose lack of the cellist in order to try to replace that along with your normal prebiotics or whatever you like to use for lactobacillus. Yeah, so one of the patients who are listeners who don't know, glyphosate was originally just a mineral chelator and they found it to have this anti-urbicite effect but the minerals that chelates is also in our gut. So when they do the studies on the cells they're like, oh, there's no problem but it has a massively profound detrimental effect to our gut because it preferentially will chelate those minerals that are lactobacillus and bifidobacter probiotics need to survive. So thanks for mentioning that because I see that all the time as well. Yeah, and it's really horrible and to try to reverse it can be difficult in a long process. So, you really have to deal with that because as long as you've got all this toxic dysbiosis in the gut, you're gonna have all this nasty stuff heading up into the liver and it's very hard to normalize your liver phase one, phase two and all your detoxification if all this nasty stuff keeps going up every time you eat. So you really have to start with the gut and that will help to reduce the inflammation in the body and lower your risk of hyperquietable responses to things. Because the lower your overall inflammation in your systemic inflammation, the easier it's gonna be for you to weather these kinds of stresses that you've come across. And again, cleaning up your extracellular matrix space that's the fluid that bathes every cell in the body with this pulsatile movement that comes from the primary respiratory mechanism eight times and 10 times a minute which is bathes and soothes every cell in the body. And the best way to do that is to make sure you have adequate cleaning up of your lymphatic fluid, of your blood, of your kidneys, your liver, if any of the drainage medicines that you prefer, clean eating is the best thing that you can do. And so these are the things, adequate antioxidants, supplements that you may need, appropriate physical exercise, keep your blood moving and stress reduction. We're all living in a time where unfortunately a lot of people are very caught in that fear cycle.  That in and of itself can create hyperquietability. So you have to learn to let go of your fear. You have to recognize, step away from this PSYOP that we're being exposed to and realize that you can have control over your life and your family's life and start taking positive steps. And I do believe that having some connection to a higher power in a spiritual way and putting your faith in that higher power and realize that good always triumphs over evil. And it's just gonna take a certain tipping point for humanity to realize that they have to choose the side of good because things are really gonna change. I love that you mentioned that because such a foundation of my practices, we know the physical and we know all these things, but these complex chronic patients that we both see, I always want to make sure that they have some sort of connection to higher power or whatever their belief system is in that there is hope and there is, because sometimes it gets so, if you're watching the news for one, it gets so heavy and so to encourage people to think on a little bit different plane can be really helpful, otherwise it's dark and dreary. You have to turn off mainstream media because most of the propaganda anyway, so you can't allow that fear driven because you're never gonna get better until you do that. So for me, many years ago, I was exposed to the practice of fallen dotha and fallen dotha is a meditation system of mind and body whereas you improve your moral character and do simple exercises you ascend spiritually. It began in 1999 in China and is spread all over the world. And so if you align with the basic truths that support all of everything in the universe, number one being truth, number two being compassion and number three being tolerance and forbearance. And the closer we align with those and the more we understand that those will always prevail over anything that's negative, then we can always have hope and ascend to a higher level. And that's the way you get better because so much of your illness is really comes from your emotional and psychological state. Yeah. So what else do we have on the, so we talked about fiber and we've talked about platelets, talked about the symptoms clinically and... See what else is I wanted to talk about. Treatment for sure. We talked just briefly about platelets, but you may want to... Yeah, again, let's talk about some of the consequences that happen when you have excessive soluble fibrin in the blood vessels and why it's such an issue. Well, if you have this sludge that's lining the blood vessel, number one, you can't get oxygen out. You also can't get nutrients out. You also can't get wastes back in for elimination and the lymphatics out in the tissues get all congested and they can't dump stuff and they get sticky. So you've got intravascular inside the blood vessels and extravascular outside the blood vessels spaces that are compromised and stuck and swollen and can't move things, it can't move toxins, it can't move nutrients, vitamins, growth factors, necessary minerals. And so you get organ and tissue compromised and that's why you have a lot of these symptomatology because especially your brain is very compromised. Also, when the vessels have that sludge, they become rigid and they can't respond to the autonomic nervous systems, constriction or dilatation orders that the autonomic nervous system is always given. So what happens when you have rigid blood vessels, you have alterations in your autonomic responses and control of blood pressure. So either these people have high blood pressure, they have difficulty controlling or they have what's called POTS, right?  So almost all of the patients that I see who've been diagnosed previously with POSCEL or the Alpha Static Tax Party Syndrome are mold patients who are hypercragable. Absolutely. Now does histamine play into the form, any sort of the coagulation cascades or any correlation with excess mast cell stuff with this or are they sort of two separate issues? I think that they are, don't ask me to tell you the biochemistry how they're connected because I don't know that but mast cell activation creates inflammation releases a tremendous amount of cytokines. That's also going to stimulate the coagulation cascade. So it's sharing too. Is exactly how is probably prostaglandin related or something because it's a perfect sense. I mean, we've been treating mast cell activation, Jill probably for years longer for anybody called it that because it was part of, it's part of the whole syndrome of inflammation that we've been seeing for years in those incredibly sensitive patients that can't take anything that'll explode anytime you try to do something with them, right? Right. The other thing that I wanted to have everyone know is that when you go to treat hypercragability you must take it low and slow because what's happened is when that fibrin has developed on the, let's put up one slide. I have, I can hit share screen. Can I view, I can do that. Let me pick what I want to use. I wanna pick a window. There we go. Okay, 22 slide 23. And I'll make sure that you have this. Okay. Now let's just, this is the way the blood's going down through it. Now this is that soluble fibrin is on the layer of the blood vessel wall, right? This is the endothelial cell here, okay? Now this is a real tight formation here of soluble fibrin and you've got all kinds of things stuck in here. So when you actually go to start releasing this all of these big bad boys like these pathogens and toxins and things down in here are gonna be released.  Also though, what can get trapped in here is thrombin. So thrombin itself can get trapped here in this fibrin sludge layer. There's no cross-link fibrin. This is all soluble fibrin but it gets locked down pretty tight. And so sometimes as you go to release this if you release it too quickly that thrombin will cause a paradoxical dramatic increase in the amount of fibrin formation that is made. So you've got to really take your time and do this slowly and just eat away at this as it depends upon how long people have been ill and whether they're, how much sort of gunk they've got and how many problems they have. Also, just to show you, this is the slide from the presentation from this Dr. Nemerson and I've adapted it from one of the lectures from Dr. Berg's permission. So here's the inside of the anethylial cell, the capillary bed. So a red blood cell is seven microns, right? So if you've got this fibrin which is only one micron here on the layer of the anethylial cell, it goes from two seconds per oxygen to go across in the absence of any fibrin to over five minutes if there's just one micron of fibrin there. So that's really a dramatic change in the time it takes for oxygen. And this is why people have pain in their muscles. This is why people can't exercise. There's a lot going on here. And this is in addition, you can stop the sharing now. You can stop, do I have to stop the sharing? There we go. Yeah, if you could stop it, I'll try it. Yeah, there we go. Okay, you got it? Yeah, perfect. Okay, we can see you again. It's another reason, not only do we have all of the mitochondrial problems because the mold toxins just stop the ribosomes and the mitochondria, the mold toxins also stop the ribosomes in all of the dividing cells that suppresses bone marrow function is all this stuff. You also have to worry about what the hyperquagability has done. So we've got all these different layers of problems that we have to deal with. When you go to detoxify somebody, say you're starting to detoxify mold, you're starting to take the toxins out of the body using the gut, getting into sweat a lot if they're able to, doing a lot of glutathione, or if you're using lipids or whatever you're doing. If you start to mobilize a lot of these stored toxins rapidly, you're going to stimulate a tremendous amount of hyperquagable response. A lot of the Hirksheimer reaction is actually people getting hyperquagable. Same thing with heavy metals. Heavy metals are just really badly toxins. And as you start to mobilize them from where they're stored, the matrix out in tissues, then it moved them through the bloodstream to the liver and out, you are gonna have a lot of issues with regulation being stimulated by those bad toxins as they're going through the bloodstream. That's why whenever you are treating Hirksheimer reactions, you need to include enzymes and not just fibrolytic enzymes, but also often proteases. Proteolytic, mm-hmm. Let's talk, we have about a good 10 minutes left. Let's maybe go a little bit to treatments and what we can do about this. This is so fascinating. And really, I'm sure the listeners and myself, and it's covering such a spectrum of our patients. I mean, I wonder if there isn't, if there is a patient that doesn't have some issue with this because it's inflammation, infection, toxin related. And I think that's all of my population. Yeah, I think almost all of my patients are on some kind of enzyme. That's the hole that I see in the vast majority of the patients that come to me from other practitioners. I agree, and I want to just again, because I'm so glad to have you here in your expertise and I have been doing this 20 years and there's many, many, many of our colleagues that don't really treat or understand this piece of it and it's critical, absolutely critical. Yeah, it really is. So there are two main fibrolytic enzymes. One is Navicinase and the other is Lumbrokinase. And unfortunately, there are many brands out there, but there are only a few that actually really work. And I don't know whether you want me to name those or not name those or tell you. Because this is all commercially free, if it doesn't matter, we can say what. All right, and I have no commercial arrangements with these companies at all, so it's not me. Really, I was taught this by Gary Klepper years ago and he's right. The Navicinase from Allergy Research Group, the soft gels, they have 36 and they have 100. I think they're discontinuing the 36, I wish they wouldn't. But there's soft. I start those people on that low dose, isn't it? Yeah, especially the kids sometimes it's bad. They, for some reason, are more effective. And I don't know what it's the way that soft gel is dissolved in the lower on in the gastrointestinal tract or not, but any of those Navicinases that come in capsules, they're just not worth their time. Now, there are some kids that have been too young to swallow even the small little football of the Navicinase. And so I'll use sometimes a combination, Natocerezyme, something like that, from like Designs for Health, or I'll use a capsule one and applesauce but use a much higher dose and hoping that I get it through the stomach, right? At least enough. Of the Lumbicinases, I'm terribly sorry, but only the Canada RNA brand named Blue Works. Agreed, totally. It's only when I blue kiss it. And I've been doing this for a long time. I started treating hypercrigibility in 2005, which is 16 years ago. I've had patients, and I've had a substantiated, I have a patient in England who had tried because it was hard to get, and she tried both the doctor's best and the algae research group, Lumbicinases. And she goes to see a good cranial osteopath. Well, each time she went off the brand named Blue and went to one of these other brands, the osteopath would say, your mechanism's all sticky. This is bad, right? So I've had other people substantiating. That's not the only example, but I've had other, you know, substantiation of the fact that, you know, that one particular Japanese made Blue is the only one that really works. So think about, well, how do they work? The narrow kinase works primarily inside blood cells. So that's gonna be really helpful now with our problems that we have with spike protein damaging anothelial cells, right? The Blue works both inside and outside the blood vessels and it's said to be significantly stronger. I don't know, there's times for me now, a kinase tends to work best and sometimes I use just Blue, sometimes I use both of them at the same time. The Blue, only about 10% of it is said to be absorbed through the gut. So I gave both of these on an empty stomach.  I rarely see anybody have any trouble with them taking them on an empty stomach. I would agree that I've had not at any issues and I have a lot of issues with gut issues, so. Yeah, and then people always wigged out, well, I don't know it, the natto kinase is derived from soy. Well, there's so little of any kind of soy protein in the natto kinase. I have not had any problems with people who are either an anaphylacid or allergic to soy, not having trouble with the natto kinase from the allergy research group. I just haven't, you know, I was worried about it and I say, well, you know, you know, I really like you just to give it a try and they just haven't had problems with it. Now, there are other seropeptivases. There are really good lots and lots of different brands of seropeptivases and things and things that you can use for hyperquietability. One that I really like is imported by Marco Pharma in Oregon is called Marcozymes, M-A-R-C-O-Z-Y-N-E-S. It's got some bromelain and other things in it, but for me, taking high doses of that several times a day for people who have been exposed to COVID, who have been exposed to vaccinated people and are worried about transmission of the spike protein to them, that shedding of the spike protein that a lot of us are worried about or the actual people who've had the vaccine itself that realize that maybe they did something they shouldn't have done. The Marcozymes seem to help to break up that spike protein as it's circulating if it's in the bloodstream. I don't have any proof of that. This is a purely clinical observation. So, and it's only one doctor's experience. So, you really have to take it with a grain of salt. But that seems to be helpful. Now, sometimes you need to use pepperon and I found that either the subcutaneous lobenox or an intravenous pepperon has always been far superior to any of the sublinguals. I know the naturopaths like the sublinguals because they don't have access in many states to injectable medications. Some states will do. Then there are a lot of other homeopathic herbs that work. Of course, I talked about the omega fats helping. They also help the blood vessels, the vitamin E, the phospholipids. The phospholipids can be incredibly healing to the blood vessels and again, really aligning. As well as certain herbs, there are herbs that help. Don Shen is a wonderful herb that helps with the coagulation issues. So, these are all things that you can use as well as the things such as a clean diet, lowering their insulin resistance, fixing their gut dysbiosis, fixing liver detoxification pathways, adequately hydrating themselves, appropriate exercising, reducing stress and all those things. Now, there are other herbs that can help. Ginkgo can sometimes help. Other anti-inflammatories and antioxidant types of herbs can help and there's so many that it's not worth trying to name them. Now, again, if the platelets are really activated, then sometimes you will need to use, I would use an 81 milligram baby aspirin a day with the high dose essential fatty acids. Now, some of the caveats to that are, someone has to undergo surgery. You really need to stop your omegas, your vitamin E and your aspirin or NSAIDs a good two weeks prior to surgery and you can restart them as soon as there's hemostasis at their surgery, like 48 hours if there's no bleeding or usually I have to listen to you the extraction, you wait till all of the oozing is brown in color and then they can restart those. I generally will stop the fibrinolidic enzymes only 48 hours before surgery and I've never had any problem with bleeding during surgery. Even if people have to go into surgery acutely and they're on these enzymes, then you just have to let the surgeon know so they may just have to hold a little longer but they'll still clot. What's really dangerous if their platelets are fully blocked by high dose NSAIDs or high dose aspirin and that's a problem. Yeah, no, I totally agree. I have not had issues and I usually just recommend patient stop before and there's no problem at all. Yeah, and they stop 48 hours before with the fibrinolidic enzymes and restart them again 48 hours later or if they have a drain, once the drain's removed or once the wisdom teeth, the drainage become brown and they really don't have any trouble at all with it. So do you keep patients on these for months or years or forever or just during the acute inflammatory phase or what kind of a timeframe are they looking at? Because I found you need them in chronic issues. They often stay on them for quite a while. They feel better. Yes, quite a while. And then if they do have one of the genetic snips, one in four people are really susceptible to mold problems. One in five are susceptible to getting hypercragable under stress of any kind. And the most common are protein S deficiencies, high lipoprotein A's, high alpha two anti-plasmids, protein C's. So if they have a significant genetic weakness which becomes obvious when you treat them, you recheck them and they still have a really low protein S, then you know that that's a genetic weakness and they may need to stay or they have a high pi one, plasma engine activation inhibitor one. They may need to stay on fibrinative enzyme to whole lives. Or they may need to always know to take them during periods of stress, any kind of stress, illness, injury, emotional stress, physical stress, those kinds of things. So it just depends. Some patients will need to always be on them in order to stay healthy. Some people will need to like, when people get a mold hit, they always have to get the enzyme because if they get over that, as quickly as they would if they don't use their enzyme. So killing the gut with things like restore and using your enzymes are as important as your things to reduce inflammatory cytokine and to bind the toxin to get them out of the body. So, you know, that's always in my mold hit protocol. So people need to understand it when to use them and that's part of what I teach my patients. Well, Dr. Corson, this has just been loaded with such great information and I just appreciate your work and being on here to get the information out. Because like I said, this is actually not common that not only our colleagues, but even patients understand. And this could be one of the most important things that we're missing. I really feel like this is so critical. And just thank you for your inquisitive nature, your curiosity, your willingness to continue to learn and grow like we all do. And just for coming here and bringing this great information today. So, so, so important. So much for asking me. I was very honored. I've been a mire of your work as well. So it's a mutual admiration society. We sort of work it, we are in the same circle. So absolutely. Yeah, and we all need each other. I always learn from my colleagues and friends with you too. You're from the same tribe. Yeah, yeah. Oh, thank you. Well, thank you so much. And everybody, I know this information has been really useful. I'll be sure and include links and link to Dr. Corson's site as well. And thank you so much for joining us on this Tuesday afternoon.


Today we're going over the Air Jordan 11 7210. But this is the low top version. Ever since I saw images leak on the internet with this sneaker, I just knew I had to have these and unfortunately, I didn't get my size. But that's okay because I'm still here to give you guys the review and I already know I could trade these for my size. And if you guys didn't know by now, my name is DJ and this is the DNA show. On this channel, I love breaking down different sneakers like this, giving you comparisons from the past and always talking about the current events as well. So if you haven't already, consider subscribing and joining the fam. We're very close to a million subscribers and you could be the next one to get us there. So with all that being said, let's go ahead and crack this box open and see what these shoes are talking about. Now, this is the Air Jordan 11 and they give us that old school nostalgic vibe with the OG 11s from back in the day. So I do like that representation when it comes to these, even though they are low top. These have an all black lift off lid. You got your Jordan air branding right here in white with the red jump man in the center. And then on the bottom of the box, it's all gray and the size tag reads Air Jordan 11 low retro, black, gym red, white sale. Ah, size nine, size nine. Usually I say size 13, just for me. But let's just get into the shoe. Lifting open the lid of the box right here, you have an all black paper and then you got the shoe. Oh, you got the shoe. Okay, first impressions of this sneaker. These are exactly what I was expecting and I am not mad at these at all. Honestly, I feel like a lot of people slept on these, but we'll get into that a little bit later in the video. Now, before I start breaking down all the styles, cut some materials of these, we got to talk about the OG pair that brought these to life. So as you can see right here, we have your modern day sleeve Air Jordan 11 box. You got your all black on the upper with the little red hitch right here. You got the red plastic with the big X in the eye, giving you that Jordan 11. Jesus thing is stiff. Giving you that Jordan 11. All right, hold on one second. There we go. All right. This thing has been in the box for a while.  Oh, you got the shoe. Now, yes, I do wish the low top version was in the same size as these. That way I can give you guys a full in-depth comparison, but I'll make sure I get some solid detail shots with the two side by side, just so you guys can see the similarities with the materials and the cuts of the sneakers. So some people may be wondering, why am I showing you the high top version if this is the low top review? And the reason why, we got to talk about the history first because nobody would like the low tops if they didn't know about the high tops in the first place. So this shoe was slated as the 7210 Air Jordan 11. Some people may or may not know, but Michael Jordan played for the Bulls and during one of those seasons, they had a 7210 record, and that set the record for the most wins in an NBA season, which was then broken by the Golden State Warriors later down the line, way later down the line. But again, that's a whole other topic. Either way, they created that based off of that storyline and called these the 7210 11. And I'm not sure if everybody knew, but there was actually a sample of this sneaker right here that was made probably about 20 or 15 years before that time in the early 2000s that wasn't even slated as the 7210s. It almost looked like a bread and a space jam mixed together. And I remember when these originally came out, some people were on the fence about them. They kind of didn't like the shoe and everybody wasn't just happy, go lucky about the 7210s. And then you obviously give it some time removed. Next thing you know, everybody loves the shoe and they want to have them and they're going up in price. You know how it typically goes. So with all that being said, throughout the video, I'm going to be breaking down the differences and similarities between these two shoes. And honestly, I feel like they did a really good job replicating the high top version into the low top version. Starting with the bottom of the shoe, you have your classic Air Jordan 11 outsole, your black pods and your creamy white translucent outsole right there.  And then you have your classic Air Jordan 11 midsole and all white and then go into the upper. All the upper. I feel like they did a really good job when it comes to this shoe overall, when it comes to just the quality of materials. I feel like that's why I say a lot of people slept on this sneaker because they're going to realize once they get these in hand, like damn, these are actually really nice. So when it comes to the patent leather on the sneaker, it has kind of that iridescent marbling kind of vibe to it. And that is very similar to the high top version as well. Then you have a nice tumbled black leather and a black suede, almost kind of like a felt type material when it comes to the eyelets. And then a little bit here on the back end. Now, this is something that a lot of people always make fun of because this is just a itty bitty, tiny Air Jordan Jump Man logo right here. But they had to find somewhere to put the branding and typically on Jordan 11 lows, they always put a little Jordan Jump Man on the back right here. And then you have your Air Jordan 23 just above that.   With your Air Jordan 11 branding in the center of the tongue and a red jump man, just like the first pair that we saw.  And this is a hit that's just like the high top version as well, super dope touch. It says, inspired by the most historic season led by the greatest player ever, AKA Michael Jordan. I'll leave the LeBron versus Michael Jordan debates down below in the comment section for you guys on that one. So overall, those are gonna be some detailed shots and some in-hand looks giving you guys a better view at this sneaker if you haven't seen these in person yet. They just came out so I could understand why people may not have seen them or if they're interested in grabbing the shoe. But now let's compare them to the high tops real quick and show you guys some more side by sides just so we can understand how similar these are to the high tops. Now obviously the boxes are gonna be a little bit different. This is more of your modern Air Jordan 11 box and this is giving you a classic retro OG style. But when it comes to the two shoes looking at the outsole's pretty similar, but when you look at the carbon fiber, way different. The carbon fiber on the high top is a lot more smooth and the carbon fiber on the low top is a lot more textured. Let me know which style you guys like more down below in the comment section. Now looking at the midsole's and the patent leathers, they honestly look very, very similar. Even the leather and the suede's and the hits on the upper as well, those all look pretty much identical. The Jordan brand branding on the tongues, very similar. Like we talked about the back of the tongues with the 7210, same exact patch. And then on the back end of the shoe, on the high top version, the 23 is stitched and then on the low top version, it's more of a plastic 23 that's kind of heat pressed on there.  And then this red on the jump man on the high top has a little bit more of a metallic sheen to it. But besides that, pretty similar. So when it comes to the highs versus lows, which one do you like more? Honestly, this is a hard one because originally I was thinking like, oh, I might like the highs more. But typically when it comes to the OG 11's and the OG colorways always go with the OG highs, but the low top, the low top just might be the wave when it comes to this particular colorway. I think I'm gonna go with the low tops on this one. Let me know what you guys think down below in the comment section. Another question that I love to ask the people in every video is if they think this shoe is fire or trash. So if you haven't already, make sure you follow me on IG so you can participate in the polls and see all the results here on the channel. This is what they said. 66% of the people chose fire and 34% of the people chose trash. I completely understand there's gonna be a lot of reasons why people don't like this shoe. It's not an OG high, it's not an OG colorway. You know, just a lot of different reasons. A lot of people wanna spend more money on different shoes. I think the retail on these was like $190 too. So $190 for a low top 11 that could potentially push people away from it too. I completely understand that. But that brings me to the next topic, the price of this shoe and what's gonna happen in the future because people always wanna know what are these gonna be worth? One, two, three, four, five years from now. If you look at the history of the Air Jordan 11 lows over the past years, you always find that same type of trend. Not that many people want them when they first came out. And then give it about a year or two and it's like, ooh, I remember those.  Like why is this shoe $350, $450? I'm so confused. Like nobody wanted those not that long ago. I think this is gonna be that prime example of that sneaker and we're gonna look back a couple years from now to see how much this shoe was going for. But I have, to me personally, I have without a doubt, I feel that these are gonna be at least $275, probably pushing mid 300s by the next couple years and then give it some more time and you never know, they might be messing around 350, 400 bucks. But again, we shall see, but I just feel like that's what's gonna happen. So let me know what you guys think down below in the comment section. And if you wanna grab this shoe, I think it's gonna be pretty easy to grab for retail or you might even be able to find a used pair for under retail market value right now. Give it a little bit of time over the next couple months and the next thing you know you got a pair in your collection to rock. But I would say, if you do have them, don't beat them up because these could potentially be some bangers down the line and you're like, damn, I'll beat them up and I wanna get another pair and I should have just took care of them a little bit better. I would say, still take care of these, you know, wear them obviously but definitely take care of these because I got a feeling about this shoe for some reason. I don't know, I feel like a lot of people are sleeping on this. Yo, if you enjoyed this video and wanna grow your collection or make extra money on the side, I built a VIP mastermind that will teach you everything that I've learned about growing my sneaker collection over the past 15 years. This will also give you access to the DNA fam in my VIP community where we talk about investing outside of sneakers. And don't worry, if you don't plan on joining the VIP community, it's okay. I'll also set up a private DNA fam community that gives you access to all the behind the scenes looks from the studio and multiple chances to win free sneakers and gear from weekly and monthly challenges. So all you need to do is click on the link down below in the description or the first link pinned in the comment section. That will get you set up and into the community. I'm excited to see you guys on the inside. If you made it to the end of this video, drop a comment down below and let me know what is your favorite Air Jordan 11 low colorway of all time. Don't forget, there's like snake skins and U.N.C.s at charity. There's a lot of cool colorway. So take a second, think about it, drop a comment. I'll see you guys in another one. Don't forget to subscribe. We're on the road to a million subscribers. Hoo-ha, yeah.