Hi friends, welcome back to my channel and welcome if you're new here. My name is Jen I'm a certified weight loss and nutrition coach and I'm on the WW or the weight watchers blue plan welcome back to my channel Happy Sunday. I'm excited. You guys are really excited for today's video But before we jump in to the exciting news today, if you're new to my channel and you haven't yet subscribed I'd love to have you here hit the subscribe button and don't forget to ring the bell right next to it So you never miss a single video give this video a thumbs up If you're as excited as I am or even if you are having a great Sunday Give this video big thumbs up and don't forget to check out the description box for everything I share with you today as well as links discounts to all my favorite things nutrition coaching I do offer personalized to you macros and calories Highly highly highly recommend as well as one-on-one coaching if you want to chat with me directly Links discounts to all of my other favorite things and my Facebook group come on over join us there as well Are all down in the description box. I can barely contain my excitement. So let's jump into today's video Like I said, I am so excited my heart is so Incredibly full last week I released the first one out of a series of four different recipe e-books that I have put my heart in soul blood sweat tears into creating for you guys and it was 15 WW friendly all plan points included calories included as well Breakfast recipes. It was a huge hit. You guys loved that e-book So many of you have reached out and said that it's been super convenient Having the option to print that out or save it to your desktop or wherever you pull your recipes from It's been really convenient for you to have them on hand All of the substitutions have been made for you. All the plan points are included including calories You guys are absolutely loving that e-book. So today I am here to announce the release of e-book number two And that is lunch recipes. This one was really really fun to put together Again, I have 15 lunch recipes for you all WW plan points included calories included These lunch recipes are my favorites. These are also your favorites What I do to pull some of these recipes is see the ones that you guys are most interested in What are the recipes that I'm getting taped in what are the recipes that you're reaching out and telling me that you've loved That I've created and I did a mixture of your favorite recipes My favorite lunch recipes and included in this e-book are some recipes You've never seen before that are exclusive to my lunch recipe e-book So let's take a look at the lunch recipe e-book starting with the cover I'm going with the same color scheme. Essentially. I'm just changing up the background a little bit changing it up So that you can differentiate between the different e-books But they all flow together really nicely if you're interested in printing them off Putting them into a binder or a recipe book to have in your kitchen I want them to look aesthetically pleasing and be pretty for you to look at when you're creating these recipes Here are some examples of the recipes in the lunch e-book. You can see it's very similar to the recipe e-book We have all of the ingredients substitutions to make them ww friendly and low calorie have already been made for you A picture of the completed meal and then if I did pull my recipe inspiration from a website I do have that recipe website noted here for you as well So we have a glaze hoisin bowl, which is one of my favorite lunches that I meal prep It's such a great meal prep. You can easily take it on the go warm it up. It's delicious. It's quick. It's easy I also am in love with this pizza pasta salad. It is so good you guys It is like pizza and pasta in one and what can be better than that And here's a little sneak peek at a pasta recipe that is new and exclusive to this recipe e-book I've never shared this on my channel and let me just tell you spoiler alert This recipe is incredible and again makes an excellent meal prep again. There are 15 Lunch recipes points for all plants calories included at the end of the e-book You'll also find links for all of the discount codes that I have for the products Shared in my e-book as well as my social media information Towards the end of the e-book similar to what you found in the recipe e-book And a big thank you at the end of the e-book as always for all of your love and support I am so excited for this e-book. I feel like as these e-books are being released I'm getting more and more excited. This one is absolutely amazing If you struggle with what to have for lunch, you can easily use these recipes for lunch or dinner They're so versatile. They're family friendly. They're kid friendly and all of the work is done for you I have modified these down to make them ww friendly and calorie friendly for you So all you have to do is get in your kitchen and create these amazing delicious meals Again, the lunch recipe e-book is $15 for all of the recipes It is available for purchase over on my website gen plate nutrition dot com Once you make the purchase the recipe e-book is sent to you in a pdf format via email That way you can save it to your computer print it off screenshot it Whatever you prefer to do with the recipes to have them handy. They are yours once they are purchased Like I said in the breakfast recipe e-book video I wanted to make sure that these recipe books were affordable It's much easier for me to create these than a big cookbook that costs a lot of money to produce As well as a lot of money for you to purchase. This just makes it super super affordable The next e-book to be released in the next few weeks is going to be dinner recipes Followed up with snacks and desserts once you have all four recipe books You have 60 recipes on hand that are tried true delicious again your favorites my favorites I'm so excited. I'm so so excited about these e-books. They've been a huge hit you guys are loving them So thank you in advance for your support. It means a lot to me I have literally poured my heart into these e-books and they turned out better than I could have ever expected And I'm so glad that you're enjoying them as well Let me know down in the comments if you purchase the recipe e-book If you're purchasing the lunch e-book and what your thoughts are again It is linked down in the description box below for you. Definitely get your hands on it It is seriously a game changer when it comes to planning prepping and preparing really good meals for you and your family If you enjoyed this video and you're excited as I am give it a big thumbs up And if you're new and you haven't yet subscribed to my channel, of course I'd love to have you hit the subscribe button and click the bell right next to it So you never miss a single video again. Thank you guys so much for your support Check out the description box down below to get your hands on this lunch e-book and I'll see you all in my next video I love you so much. Bye friends


In the last 3 modules we have discussed femtosecond up conversion spectroscopy, femtosecond fluorescence up conversion or femtosecond optical gating fog. Today we have come to the lab and we are going to show you an actual fog spectrometer and you will see how the data is recorded. But before we do that let us recapitulate what we have done in the class. I will first show you a diagram of the system then we will go to the instrument. You might remember that this is the layout of our optical unit for the fog spectrometer. The spectrometer we use in our lab has the name fog 100. It is from a company called CDP Corporation which is based in Russia. So just to remind you what we have here is that we have red light nominally 800 nanometer but in our case tunable from 690 nanometer all the way to 950 nanometer from a femtosecond pulsed titanium sapphire laser. Later on in this course we are going to open up the laser that is used as a light source for this instrument and we will show you what is in there. Let that be the story for another day and let us wait until we discuss the operation of a titanium sapphire laser in class before we come back and show you the laser. For today we just take the laser as a light source 100 femtosecond pulses 80 MHz repetition rate. This light source passes through an aperture and is focused by this lens L1 onto a non-linear crystal NC1. Here we typically use a beta barium borate BBO crystal and by focusing the red light onto the non-linear crystal 1 second harmonic generation takes place and you generate blue light. Now in this region you have red and blue light that is that are mixed together. Since you focused you are going to have a diverging beam here the diverging beam is captured by another lens L2 which is placed in such a way that the focus of L2 is at NC1. Then the mixture of red and blue light goes and hits a beam splitter BS1. Here red light goes through and blue light gets reflected through something called a Berek wave plate that we are going to show you blue light goes and hits another beam splitter through which any residual red light is dumped and the light blue light itself is reflected. This light is focused by another lens L3 onto the sample and as you are going to see the sample that we use is mostly liquid sample which we rotate all the time so that it is not destroyed by these femtosecond pulses. We are also going to learn later on how much energy per pulse is there. Then from the sample fluorescence is collected by another lens goes through a long pass filter F3 which blocks the excitation blue light but passes the fluorescence light. This fluorescence light is focused by a lens L4 onto another non-linear crystal which we called NC2 or as we have discussed in class the sum frequency generation crystal. So this is one part of the story. The other part of the story is that the red light that passes through BS1 hits a mirror M2 another mirror M3 and then gets retro reflected to a mirror M4 to M5 from M5 it is focused by the same lens as the one that is used to focus fluorescence light onto the same non-linear crystal NC2. So here what you have is a sum frequency generation you remember this is called gate light the red light. Let us say that has omega 2 frequency let us say omega 1 is the frequency of the fluorescence. I do not exactly remember whether you use omega 1 or omega 2 but I think you can understand so what happens here is that omega 1 plus omega 2 sum frequency generation takes place and it is this sum frequency that is made to go through another filter which in this case is a short pass filter passes only UV and we have calculated in class how UV is generated here and this UV goes to a monochromator and then to a detector. So the way it works is that this retro reflector is mounted on an optical delay optical delay is essentially a one foot long screw which is moved. So the screw is moved to a particular position then intensity of sum frequency is recorded then it is moved to another position intensity of sum frequency is recorded and this is how the entire map of fluorescence decay is generated by plotting intensity of sum frequency against the delay time and as you might remember we had said that since the intensity of the gate pulse is constant and intensity of fluorescence falls off with time intensity of the sum frequency it is product of intensities of omega 1 and omega 2 the intensity of sum frequency actually provides a measure of intensity of fluorescence light because intensity of gate light is constant. So when we plot that against delay time we generate the fluorescence decay with femtosecond time resolution and while calculating this time resolution at goofed of a little bit because I had taken frequency of the velocity of light to be 3 into 10 to the power 10 meters per second actually it is 3 into 10 to the power 8 meters per second. So flux of the matter is if you move the screw in with microsecond resolution then you can obtain a time resolution in femtoseconds. So that is a recap that we wanted to provide now let us go and see the spectrometer alright this is the optical table on which our femtosecond optical gating experiment is done this here is the femtosecond laser in our case it is a tsunami laser from Newport it is quite old the other laser that we have not shown you yet I think is much compact but what tsunami does is that it provides 800 nanometer light with 100 femtosecond pulses at 80 megahertz repetition rate. So that light you can hardly see it comes here hits this mirror comes here goes through this optical unit which we are not going to discuss because we are not talking about third harmonic generation from there it goes into the spectrometer this is the spectrometer for 100 where the experiment is actually done and for your benefit what I will do is I will remove all this so that you can see a little better this here is the optical unit remember where laser light is supposed to come from it is supposed to come from this direction now let me show you the red light if you can see it you see the red spot of light that is the output of the titanium sapphire laser to your eye it seems that it is a light that is always on but actually it is not we have 100 femtosecond pulses there and the pulses are coming at a repetition rate of 76 megahertz too fast for our eyes to make out the pulses from each other that is why it looks like continuous wave. So this comes here this is the lens that for dramatic effect let me do something else let me block this or maybe not this is the lens that focuses it onto that is better focuses it onto this non-linear crystal and you can already see some blue light coming out from here right as you see the intensity of blue light is something that depends on how I turn the crystal you see the blue light got brighter and if I turn the other way it is going to get dimmer and dimmer. So these crystals are angle tuned as we are going to study perhaps later on you can actually change the angle of the crystal by using this micrometer screw head and that is what is going to affect the efficiency with which second harmonic is generated. Now this light you can only see blue light here because it is so bright and our eyes do not see red light all that well so this blue but actually it is a mixture of red and blue so that comes here onto the beam splitter this is the beam splitter so if I hold the card here you can see the red beam once again that is because the beam splitter has reflected most of the blue light and has transmitted the red light I have always been talking about the blue path first so for a change let me talk about the red path to start with. So remember this is the gate light the red light it hits the mirror M2 M3 and go state this here is a retro reflector you can see the spot here this is a retro reflector and this thing that you see and you see it better when we turn the lights on this is a retro reflector remember and it is mounted on a one foot long screw you cannot see the screw because it is inside when we show you pump room later on you will actually be able to see it and what this retro reflector does is that this retro reflector can move all the way from here to here we will just move it once and show you see this is computer control your TA just click the mouse and now you see the retro reflector is moving back and as you understand while it moves back this red light here is undergoing a longer path so this is how we can actually change the path length of the red light which as you know by now is called the gate light so right now we are going continually just to show you but actually we go in steps we go step by step and then we stop at every step and that is where the measurement is made okay so you have seen the screw now let us get back to the demonstration of the light path we have taken the retro reflector back here this is the incoming beam and the outgoing beam comes from here onto this mirror I think you can see the spot on the card here so it hits this mirror here this mirror if you remember is called m4 this is m2 this is m3 this is the retro reflector R this is m4 from m4 it comes to this mirror m5 and from m5 it goes to this lens which focuses it onto this crystal this crystal is the nc2 crystal the second nonlinear crystal they stand on the light so that you see the components once so to repeat this one is m2 m3 retro reflector R m4 m5 this is a lens l4 which focuses the beam onto this nonlinear crystal nc2 and from there the output is collected by this lens here this lens is l5 go straight you see this is opened all the way this piece of optic here is a short pass filter it allows UV light to go through but not visible light so you understand this is the path of this some frequency as well as we will show you this here is the monochromator as I told you this is an old machine so it has developed some light leak so you have covered it with black cloth and this is what contains the photomultiplier detector so what we have showed you now is the path of the gate light now let us talk about fluorescent light I show you the path first while the light is on then we will switch the light off and show you the light well they show you the fluorescence so now remember this is beam splitter red light has gone through and blue light has been reflected in the path of blue light we use these pieces of optics which are called neutral density filter the reason why we use them is that you do not want to put too much of sample on too much of light on your sample anyway it will get destroyed so from there blue light comes in this thing you see is called Berek wave plate because as you remember you have to maintain magic angle polarization if you are wrong there your decays are not going to be correct so this is what is used to measure to maintain polarization this is a second beam splitter which reflects blue beam and transmits the red beam the blue beam is collected by this lens inside the cylinder there is a lens and is focused on this sample the sample we have is a liquid sample so what we have to do is we have to keep rotating it so that the laser light does not hit the same spot again and again because if it does then your sample is going to get spoiled fluorescence from there is collected by a lens kept inside this cylinder go straight and passes through this filter which is a long pass filter cuts out blue light and transmits fluorescence light then this fluorescence light falls on the lens l4 you might remember l4 from our previous discussion of path of the red light l4 focuses the fluorescence light and remember as well as the gate light on to nc2 the nonlinear crystal some frequency is generated here collected by the lens here goes straight and we have discussed the detection part already so this these are the components now we are going to turn off the light and show you the fluorescence as and as well as some frequency right now that the light is off you can see blue light once again can't you blue light comes here remember it is collected by this lens and is focused on to the sample the moment I remove this barrier you will see the fluorescence of the sample yourself and that is because right now you are using a standard sample which is very highly fluorescent there you go the bright light that you can see is that of the fluorescence and from this side if you look you can actually see a spot that is brightest that spot is where the blue light is focused and that is why the fluorescence from that spot is what we are actually collecting the everything else is a glow arising from there now remember the path once again the fluorescence is collected by this lens goes straight if I put in my card here you can see blue light as well as the fluorescence now we don't want blue light that is why it goes through this long pass filter as we have discussed and after the filter if you put in the card you see now there is no blue light you see that you put it here you see a blue sharp spot almost at the center of the spot that you see for fluorescence when it goes to the long pass filter that long pass filter does not transmit the blue light anymore but fluorescence goes through and here fluorescence looks like a big spot size of which is determined by the diameter of this lens then what happens it goes here and on this lens l4 now you can see both you can see the sharp red light which is gate light and you can see the fluorescence light as well as a big spot both are focused now now if you see if I go more and more towards NC you can see the spots are coming together and they are becoming smaller they are becoming smaller because they are getting focused and they focused on this non-linear crystal now what I have done is I have intentionally detuned the crystal here to show you what happens after the crystal okay let's go back once before the crystal you can see the gate light is towards me and the blue light is away from me or from the direction in which you are looking red light is towards your red towards your right and the fluorescence light is towards your left after the non-linear crystal the directions have reversed because they have crossed here so the in a perfect alignment they would have met exactly at the non-linear crystal now what I want to do is I want to generate some frequency to do that I have to angle tune this crystal and you'll see some frequency coming out as blue light actually it is UV but when it hits the paper it looks blue yeah there you go in fact you can tell by I by enlarge and but later you have to use the detector you see you see this sharp blue light coming out that's actually not blue it is UV but UV light excites the collagen molecules in paper and that is what gives you a blue color to your eyes now it is this light that goes through here of course you can see everything but if I could put the card after the short pass filter you'd see fluorescence is cut out gate light is cut out only this UV light which shows up as a bright blue light that is the only thing that goes through to the detector right now we are going to acquire data you see our computer screen and there I'll draw your attention to the panel on the top left there you can see two readings on top you see something like 28,998.84 femtosecond that is the delay that we have given to the gate light it looks like a ridiculously long large number but that is because we are writing it in femtosecond if you wrote it in preco second it would not look as bad but then the point is that we actually have this kind of accuracy that we can go to the second place of decimal of femtosecond and the bottom panel where you see a number that is fluctuating slowly you saw 350 now it is 373 little later it will be something else 415 that is the output of the photomultiplier tube and that number is proportional to the intensity of the sum frequency light and we are going to show you first of course you cannot see me now but I'll tell you what I'm doing first of all I'm going to block the fluorescence light so see right now count is something like 364 or something now I block the fluorescence light and immediately the count starts falling now it's 292 now it's 60 so it's going to go to almost 0 and we are using a slow acquisition time that's why you still see some counts otherwise you see nothing so the count becomes almost 0 when you block the fluorescence light that's one part of the story now I have unblocked fluorescence light and you can see the count growing again after 2 or 3 readings since we are using a 5 picosecond 5 second integration time it takes a little bit of time but we are back to where we started from 358 normally this count is much higher right now our instrument is not in very good alignment normally this count would be something like 80,000 or so now next I'm going to block the red light I've blocked the red light now and you'll see this count falling once again see it started falling already 294 from 300 something now it's 69 so what does that mean the signal goes when you block red light or you when you block fluorescence light this is what confirms that it is actually some frequency signal because if you block one light and the signal doesn't go that means it is some kind of a spurious signal next what we are going to do is we are going to record a decay which means we are going to change the delay and we are going to record the intensity of some frequency as a function of delay time so now see we are recording the data and right now the time delay is such that we have not reached time 0 you might be able to see that the data is being recorded point by point and you can see some fluctuation in data that is the noise that we had talked about the data you don't get the same value for all measurements even when the counts are actually close to 0 so at time 0 what happens is there is a jump and the decay the signal goes up and it decays from there now you see that there has been a jump which means that we are now close to time 0 and what was looking like signal to you a little earlier you can see now that it is actually nothing but baseline it is 0 you might be a little confused by the way acquisition is being done here the way this program is written is that the x axis keeps changing and keeps getting expanded as you record so that is why what look like full scale now is no longer full scale it's getting actually smaller and smaller now we have reached time 0 and that means that signal is actually there and you see that there is a jump now you are going to see the decay of the signal in fact for the sample that we have it does not decay too much it actually has a nanosecond lifetime but this whatever much it decays in our time of acquisition you are going to see it here in fact now that we have shown you time 0 what we will do is we will stop this acquisition and we show you a data that we have recorded previously and you should be able to correlate that with this one right so this is what you see here is a decay that is recorded by the method that you saw y axis is actually intensity of some frequency x axis is delay so this as we have said earlier is essentially a map of the fluorescence decay that the sample has so to conclude this discussion we have discussed in the lecture how fluorescence upconversion technique gives you a femtosecond time resolution and here today we have given you some glimpses of how the experiment is actually done so the only thing that remains for you to is to go to a lab and do the experiment yourself we conclude this part of the discussion now and later on after this we are going to move on to a discussion of how lasers work because as you understand lasers are the central tool for any kind of ultrafast spectroscopy that we do and once we are done with the discussion of the theory of lasers we'll come back to this lab we'll open up the laser for you and we'll show you what is there inside


Welcome to Healthy Planet, the show for people who care about their health and the health of our planet on the ThinkTech Livestreamy Network series. I'm your host, Dr. Grace O'Neill. Joining me today is Dr. John McDougal. Welcome Dr. McDougal. Tell us about your journey to discovering the starch-based diet and how it helps people with chronic disease. Well, thank you for having me on. My name is John McDougal and my beginning is in Hawaii. I don't know whether you can see the full shot there. But if you do, you see the picture of the Hawaiian Islands behind me because I really have my experience in Hawaii to credit basically everybody, everything I am today. I was raised in Michigan and wanted to escape and decided the nicest place in the world to escape would be the Hawaiian Islands. And so Mary and I left there for there in 1972. Did a internship at Queen's Medical Center. And then I became what is known as a sugar plantation doctor. But you know, people don't even recognize that particular entity, sugar plantation doctors. I've asked them, you know, do you remember the sugar plantation? They don't. But anyway, I was a sugar plantation doctor, which was my job along with three other doctors was to take care of all the employees of Hamakua sugar, which were 5,000 employees.  We were it as far as the doctors are concerned. Well, what I learned was two major things. One is that I wasn't taught how to treat chronic disease. As a result, essentially all of my patients with chronic conditions stayed sick and continued to get worse. And I thought I was a bad doctor to tell you the truth. I kind of blame it on myself if they weren't getting better. So people with high blood pressure, diabetes, cancer, heart disease, arthritis, and so on, they never get better.  It was the first thing I learned is just what shortcomings medicine has for the patient. The second thing I learned was how important diet is. And I think the big on the whole, I was unique in the sense that we had this population of people that were first, second, third and fourth generation. Our first generation were people from Japan, the Philippines, China, Korea, who learned their native diet while they lived in their native lands. They learned a diet of rice and vegetables, and they brought that diet with them when they moved to Hawaii to start new families. Their children were a bit more influenced by the Western diet. And as a matter of fact, right up from our our plantation was Texas Drive-In, the home of the Malasauva. And lots of people ate there. In fact, you don't go to the big island without eating there. So they ate more rich food, more greasy food, more meat, more dairy, et cetera. By the time I got to the third generation, I was taking care of people who were just as sick, just as overweight, as people I'd learned about medicine in Michigan and on the island of Oahu. And it was because of their food. They lived on rice. That's why they were healthy. And since then, I've spent my whole life, the last 47 years, taking care of people with that observation in mind. That is rich foods make you sick. And the right diet for people is a starch-based diet, like my Asian patients ate. This one was based on rice. If you take people who are sick, obesity, high blood pressure, stomach trouble, diabetes, et cetera, and you put them back on the diet for human beings, they do well. And so that's all I've been doing for 47 years. Taking care of 12,000 people. Myself ran a couple of live-in programs. One was based at a hospital. One was based at a resort. And right now we run a 12-day internet-based program, which is our most successful attempt at helping people. We take care of them over the internet for a 12-day period of time, medical care, et cetera. Anyway, I've had a very rewarding practice for the last, what's been going on more than a half a century. Yes, you've helped a lot of people and helped a lot of people also lose weight. I've been listening to your book on Audible, the McDougal program for maximum weight loss. What do you recommend if somebody is in the same vein as your book, I guess, trying to lose a lot of weight and they're already plant-based, like you recommend avoiding certain things, correct? What would those things be? They'll plant-based or vegetarian or even vegan doesn't really mean anything in terms of what I consider good health. You can be a vegan and live on coaxing potato chips. You know, vegetarians often, they think that, well, vegans, including vegans, they think fake foods and lots of oils are just fine for them. The vegetarians, they'll include dairy and eggs in their diet. You know, you're not talking about anything that resembles good health. What we're talking about is a diet based on starch. If you do a little bit of history and you look back at what people have eaten, you find that 99.999% of people who ever walked this earth lived on starch-based diets. Native Hawaiians, they lived on taro and breadfruit. That was their diet. It was only when the missionaries came and brought the pigs did they end up eating a lot of rich food. It was the queens and the kings that were at Hawaii who, these royalty and their fellow aristocrats, they got to eat the rich food. But otherwise, the traditional Hawaiian diet is a diet of starch, of taro and breadfruit with, you know, a little fish on occasion when they caught fish, but it wasn't the dominant part of their diet. And so we can go, we can go all over the world and make the same analogy. You go to Central America, Mexico, these are the people of the corn. They lived on corn. The Native American lived on, I mean, we're talking about 12,000 years ago, the Native American lived on diets of potatoes and sometimes diets of corn. You go to South America, the Incas lived on potatoes. Go to the bread basket of the world. You know what that is. That's what they're talking about every night on the news. Iran, Egypt, Iraq, Ukraine, the bread basket of the world. This is not the pork chop basket of the world. This is the bread basket of the world. And we just started out talking about what Asians eat. Typically, you know, that's what most people think that Asians live on. But Asians have lived on many, many other starches successfully, not just rice. So you also said people should minimize, like say somebody is based, they should also minimize their avocados, nuts and seeds as well. And soy, I think was also something you said that people should minimize if they're trying to lose weight. If you want to lose weight, you got to do. It's not that they're unhealthy, nuts and seeds and avocados. I would consider them healthy. But they're very rich.  So whereas, whereas, you know, back when, when an avocado tree only came in bloom as a dozen Hawaii, you know, for a week or two weeks, now we have avocado trees in bloom all year long at, at Safeway and Kroger and other markets. And that's the problem. You know, we're nuts. You know, nuts provided some extra calories for people to store as they were going through a difficult winter when crop wasn't available. So you have nuts coming in season again at the end of summer, the beginning of fall, which will provide some extra concentrated calories to get through through a difficult winter. But otherwise, you know, starch has been the diet of essentially everybody that's walked this earth with a few exceptions. The exceptions being the aristocrats of old who were obese, had diabetes, heart disease, gout, et cetera. And the other exception would be the few populations that have lived at the extremes of the environment like the Inuit Eskimo. But otherwise it starts to power people throughout history. And you know, that's, that's what the human being eats. And the reason people are sick. And I saw it. I mean, good grief. I was there for 15 years in Hawaii. And I saw the transition in health and people, you know, where, you know, predominantly the Asian population was trim and healthy, but not, not anymore. You know, and the sickness, I mean, some of the statistics, and I don't want to quote them because, you know, some of them may be a little dated, but as I recall the Filipino has the highest rate of gout of anybody on this planet. And the Chinese has the highest rates of cancer. The ones that live in Hawaii, I'm talking about. Yeah, yeah. You know, we have an epidemic of diseases that are due to eating rich food. And the simple solution is to go back your, your ancestors diet. Your grandma, or great grandma, and they have to go that far. When you asked for dinner, what was for dinner on Sunday? You know, she said rice or potatoes. Now, when you ask for what's for dinner, when you go over to visit on Sunday, it's pork chops, you know, or steak or fried chicken or whatever.  And for the previous main years, they eat starch-based diets. This is just an aberration, the rich Western diet is caused the dominant amount of sickness, the heart disease, you know, major organ cancers, diabetes. Anyway, it goes on and on. And plus, look, when 80% of the population, and it may be worse in Hawaii, when 80% of the population is either overweight or obese, something wrong. It's not people. It's something terribly wrong. It's not a lack of medications or treatments. It's the food. That's what's wrong. So what do you think about people who are concerned about getting adequate omega-3s on a plant-based diet? What would you say to them? I would, I would ask them, what makes omega-3s? And they'd say fish and I'd say no. No animal makes omega-3 fats. Can't be done. Never has been. Never will be. No fish ever made an omega-3 fat. Well, where do you get omega-3 fats from? Do you have a plant? No. Why does a fish have a lot of omega-3 fat? Because the fish eats seaweed and algae and stores it in its body fat. So why not go to the original source of omega-3s? You'll never miss. There's never been a case of omega-3 fatty acid deficiency on any natural diet. It doesn't exist. Something made up to cell supplements. So you don't believe in the ratios that people are are recommending, you know, there's a certain ratio they recommend to omega-3 to omega-6 ratio. Whatever the ratio is, it's what plants have.  You know, if we were, and I read an article today about the carnivore diet, which is where people are living basically on just only animal food.  You know, you don't have to wait long on an all carnivore diet to get into trouble. In fact, one of the examples was when Robert Atkins started out. Bacon, butter, and brie was his diet. No plants, no carbohydrates, and of course Robert Atkins died obese with terrible heart disease. He was a sickly man. Do you remember that I was when he died? I ought to live with Robert Atkins, so that might say something to you. So what would you say, a lot of people are concerned about having this double copy of ApoE4 gene, and they want to prevent Alzheimer's. What would you say would be the best person a person like, what would be the best thing a person like that could do to prevent Alzheimer's? I can't use your genes, can you? So that was out, and it's only an association, it's not a cause-and-effect issue. Alzheimer's is not a genetic inherited disease due to food poisoning. It's due to one, excuse me, not just food poisoning, but one particular specific element that's commonly found in our food. The first case of Alzheimer's disease was discovered in 1903 by Lewis Alzheimer. First case, and now it affects millions of people worldwide. I'll give you the short answer, and you can look it up. You can study it, you can spend weeks studying it. You'll come to the same conclusion that I've come to, is Alzheimer's is due to aluminum poisoning. The metal aluminum is Alzheimer's, and so that's why it's epidemic. That's why it's not likely to be solved soon, because the aluminum industry is very, very powerful and rich. They have a lot of lobbyists who keep the trick. Keep the secret a secret. So we have a pretty good question from one of our viewers. The question was, what if a person ate a healthy plant-based diet and exercised, but still came down with a serious illness and condition anyway? What should that person do? Should they change anything in particular? Well, what if you had a race horse, and a race horse broke its leg? Would you put it on dog food? No, of course not. Okay, so there are problems that happen besides those due to food. Those caused by food. You can get hit by a car. You can catch an infection. You know, there are lots of things that happen to people. The vast majority of illnesses are due to being a glutton, to eating the richest diet known to human beings.  That's the human diet with the addition of fruits and vegetables.  They ate anything they could get their hands on, including I was just watching a report on how Austrians used to live on grubs, you know, little worms? Yeah. Grub. So, you know, right, you know, it's not a vegan diet we're talking about. It's talking about a diet based on starch. And when people ask, my patients ask, you know, how much starch should I eat, doc? I say, you look at your plate, 90% of your plate should make up the amount of starch. And then the rest of fruits and vegetables. How much animal food should I eat? Well, you need none.  Two issues there. If you've been to Europe, and Europe ate the salad last. The second issue is, do you know anybody with deficiency diseases? Do you have friends with scurvy? No. Berry, berry. Allegra. Why would you be looking for more nutrients when it's not a problem? The problem is in excess. Do you have friends that suffer from excess fat? Yeah. Excess cholesterol. Excess salt. You know, a problem is a problem of excess, not a problem of deficiency. Why look at the wrong direction? Why? Because lots of people get to sell lots of supplements and make billions of dollars. That's why. It's just money. Now, how about somebody with hypothyroidism? Do you have any thoughts? There are some plant-based people and they have hypothyroidism. Is there anything they could do for that? They could take thyroid supplementation. They have to. But there's nothing in terms of their diet, like would you advise them to avoid differences or soy or something like that? The thyroid gland is dead. In most cases of thyroid deficiency hypothyroidism, your doctor will tell you are due to autoimmune thyroiditis. In other words, the body attacks the thyroid gland. And the reason the body attacks the thyroid gland is because we eat foreign thyroids. We eat thyroids from pigs and cows. Do you know how we eat foreign thyroids? Do you know where you find them on your dinner plate? In animal products, it's definitely in the animal products. Like hot dogs or something. Hot dogs. Yeah.  The body recognizes them as being foreign. And it gets mixed up and attacks your own thyroid gland. It's dead. It's not a grow back. I don't care what you do. But you can live healthfully, inexpensively, safely by taking a thyroid supplement, which is what my patients do. I'm a real doctor. I'm a board-certified internist. I've been practicing medicine for half a century. Good grief. I don't practice anything that is other than what it should be, other than proper care of people. And I don't treat dietary diseases with drugs. Yeah. Except as a last resort. I may deal with some of their issues. But you take dietary diseases just by fixing the food. It's like when somebody has a bad cough and they have mucus coming out of their lungs. You could give them a cough, sir. Or you could tell them to stop smoking. Yeah. Yeah.  Or I could tell you to stop eating the foods that are making you sick. And those are the animal foods and the free oils. Yeah. You know, like flaxseed oil or corn oil and safflower oil. These are toxic. So once you do that, what happens is you heal. Well, our results are we get nearly 90% of people off of their medications, particularly their blood pressure and diabetes medication. 90%. You know, our weight loss is like 3.1 pounds in seven days. It's 20 pounds on average in a year. Nobody else gets those kind of weight loss results. You know, we have, you know, our curate for, well, it's really high. Is that the in-person program? Or are you talking the- Oh, we've done it, we've done it several ways. I did it for 16 years at St. Helena Hospital in California. I did it for 18 years at a resort in Santa Rosa. And then with the pandemic, everything closed down. And so we brought the program to the internet, which is the best thing that ever happened to you and to us. Patients get so much more out of an internet experience. We do telemedicine. We come into your home several times a day. Lectures are good to your convenience. We're running on right now. We have real 50 happy people who we're taking care of, and they come from Sydney, Australia, and Shanghai, and London, and they're all over the world. That's great. I started practicing. I started, my first practice was for 15 years. After I finished my surgical internship, I did a residency at John Byrne School of Medicine for Internal Medicine. Then I practiced as a general practitioner the rest of the time I was there, which was a while, maybe another eight years or so. My kids are full Hawaiian. They were all born in Hawaii. But Mary and I are about as Hawaiian as you can get being transplants. That's what we think of as our home. That's why I have the picture up here. Do you see the picture? I show that to everybody that comes into my home for an interview. What was I going to say? Is there anything that you would tell in terms of diet for an 80-year-old to eat differently than a 30-year-old? Are there nutrients that they should get more of?  I don't know. I think things have passed a lot. An observation that I made when I lived in Hawaii, hopefully you can still make today, that that is that grandma and grandpa who still maintain their starch-based diet, whether it be rice for an Asian, whether it be taro and bread food, potatoes for your native Hawaiian, they're the ones that are still working in the yard, are still trim and healthy. Their kids are sick and obese. What happened? Did their genes change?  They changed their food. That's what got me so excited about it. I saw this right from my eyes. You can definitely see it. I used to work at Kuakini and you can definitely see it because there's very, very old patients and then you have their grandson or granddaughter that comes in and you can see through the generations what people look like when they arrive in Hawaii. Why did they look that way?  They look that way because they lived on starch, which means also too much fat and too many animals and too much cholesterol and too much protein, but you've got to get that word starch in your vocabulary. You're going to be out of control. It's just like the good morning greeting for Chinese instead of saying, hello, how are you? They say, have you had your rice today? And a bride is not considered a proper bride until she properly knows how to cook rice. Look, it's not been that far that you've come from your beginnings from your grandparents. Maybe your parents, you can still see this, but you better look fast because it's going quickly all over the world. My parents just came from Taiwan, but I was born here. So I do want to go to your website though because I think there's a lot of good resources. So if you could go, Eric, to his website and show people who are watching online. There's a lot of things here. I was actually going to try out some of your recipes. They look really good because I was surfing your website. This one of the vegetable chowder, that looks pretty good. There's a lot of ones that look good. They have beans and rice. And then if you could show his program. And by the way, we also have a free program. We have the program where our staff, our team takes care of you and helps you get off your drugs or on the right drugs. We have that program, which is our main effort. But Mary and I, since we've had a website, we've offered the program free. It's on the website, 12 days of menus, all the instructions, all the recipes you'd ever need. A good share of the medical advice you could ever use is on that website. Yeah, the website's great. Sometimes I Google for my patients actually about different problems people have like kidney stones, which is very diet related. There's a lot of things you can do for your diet to decrease your incidence of kidney stones. And I've taken some stuff from your website because it's been very helpful. And I think there's some good information about gallstones as well on your website that I've given to patients. So I find it very helpful as well. If you can be an adversary to teach the kind of diet that your grandma and grandpa ate, then you're going to do just fine. That's the problem. And that's one of the problems with this kind of eating. In addition to the fact that it's hard to make a living, there aren't very many businesses that come out of feeding your potatoes, is that a person with a seventh grade education can teach you. You don't have to go to medical school to learn this. So if the common person could share this profound message of healing, it shows you just how simple and easy to understand it is. The truth is simple and easy to understand.  They eat rich food. The way you get your health back is eat the human diet, which is a starch-based diet with fruits and vegetables. That's it.  Well, thank you for all the work you do. All the work you've done all these years and your books are fabulous. I don't know if you can show Eric the starch solution. And then I've also been listening on Audible to the McDougal program for maximum weight loss. Both are fabulous books and you go through actually different nutrients that people might be concerned about like protein, calcium, when they change to a plant-based diet. And I think that's very helpful for people as well. I didn't really get to go into that. But thank you so much for being on the show. We do a free open talk to the public every Sunday night at 5 p.m. Pacific time. It's on our YouTube channel, which has got loads of educational videos on there. But we also do this time, just like you and I spent the last few minutes together, a time where people can ask us questions, no charge, no obligation. You don't even have to identify yourself. And so we offer that to the public. Sunday night, Pacific time. Wonderful. Well, thank you so much for being on the show. We're out of time, so we have to wrap it up. I'm Dr. Grace O'Neill. This is Healthy Planet on the ThinkTech Live Streaming Network series. We've been talking with Dr. John Doogle. Thank you, Eric, our broadcast engineer and the rest of the crew at ThinkTech for hosting our show. And thanks to you, our listeners for listening. I'll see you in two weeks for more of Healthy Planet on ThinkTech, the show for people who care about their health and the health of our planet. My special guest will be Winston Walsh of the Manoa Outdoor Circle. We will be talking about protecting and preserving Hawaii's environment. If you have ideas for the show or questions for my future show guests, please contact me at HealthyPlanetThinkTech at gmail.com. Check out my website at graceandhawai.com or Instagram at GracefulLiving365 for more information on my projects, including future show guests. I'm Dr. Grace O'Neill. Aloha, everyone. Thank you so much for watching ThinkTech Hawaii. If you like what we do, please like us and click the subscribe button on YouTube and the follow button on Vimeo. You can also follow us on Facebook, Instagram and LinkedIn and donate to us at ThinkTechHawaii.com. Mahalo.


What's up everybody Jason here from jazby's case breaks calm We just filled up 2020 tops gold label baseball 16 box case break picker teams number eight So there you go Gold label baseball 16 box case break picker team number eight All those character signs meaning we did a little filler that 21 teams in the filler guys There you go There you go Roger got a spot on the break again has to fill by time. I'm done with this guys Fill it up fill it up and the metal for stripping on the lot with my last break anyway, so Do this one or the last one over there? All right guys Juan got the Pittsburgh Pirates I think one of the boxes either has a redemption in there or It was missing a hit, but I've seen other boxes in a master case where it has the relic and an auto so It might be on the other side, but just what I why I don't know Nick I mean he was jogging on the sideline After that injury which I didn't even know he got injured, but I don't think it's that serious I think it's his ankle. I think his ankle might have got like a little rolled or bent back a little bit, but I Don't think there's I don't think it's too serious, but I guess we'll find out tomorrow and for Thursday night status because He was jogging up and down the sideline. They said and I guess maybe he just couldn't do it like Or at the time they part that hey man this game's out of reach so who knows They don't really say too much until like after Yeah, I well someone that said his knee, but then they said it was an ankle But I follow that pro football doc on Twitter and he said that well, I shouldn't be an ACR like that. So Could be either Something to do with his meniscus or like they said later. They were little to find out But dude man, like he's like he missed week one and so far he leaves He's like the dirt. He started rushing Isn't the crazy stat my Sanders is third in rushing yards in the league and he missed the first week of the season But he's 16th in carries per game So he's right in the middle He's like he's like taking off and exploding for these like huge runs and not really getting the ball Enough carries a game Well, it's like dude, you guys just need to feed him But at the same time I understand why they're not feeding him because the line is just totally trash They've already been on their third stringer Unlike red guard and left tackle The only healthy starter on the line that's been healthy all year has been Jason Kelsey They were on their fourth string tight end today. He caught a touchdown pass. I didn't even know who the guy was Cal Lewis Cal Ruben junior No synagogue All right, so I'm just gonna go through all of these base cards First and then we'll go through the hits after that's okay trade turn into 75 Just get the bigger rookies and numbered cards out of here first Louise Robert, that's class one Nico JD Martinez All right, so this is the one that either there's a redemption in here or it's just missing a hit. Okay. There is a redemption right here. Oh That could be that could be Louise Robert Robbin can oh That's right Oliver. He is He is yes, sir Walker Bueller.  All right, Tyler. Yeah, I mean, I'm sure there's other players that are redemptions too, but At least you kind of already know okay if it's a redemption. It could be this person George Springer Johann Moncada Gavin Luxe Yeah, did I miss them already? I definitely unless I miss them Trout to 150 I've been looking at all the RCs and only doing the bigger players of course Kesta, I pulled in a Rosarino the other day in a personal box. It was pretty nice a framed autograph Love to pull in here. Willie Mays to 99 Gavin Luxe 15 out of 50 Louise Robert class two. All right, let's go with the frame daughter guys. Good luck guys There's a Louise Robert class three on the top. That's pretty cool And we got Dale Murphy to start us off for the Braves. Very nice Braves. It's going to Matt. The next one Anthony K for Toronto Robert with the blue jays skip the redemption. It's making more suspenseful than it already is Ranghet Rangel Revello for St. Louis runner Robert Nick Solak for Texas going to Todd. I got three more Plus the redemption for four laps. I got a blue Wow, it's Ronald Acuna, Jr. And I want to say Matt I Want to say that you told Joe this past week when we got this that you didn't really hit too many Braves So if y'all don't know if you're watching live with this But I want to say that that day you bought the Braves and a lot of these and went hitless That is the case not no more. You got two hits in here including a Ronald Acuna, Jr. 38 out of 50 Very nice Carlos and at a 75 Michael broselle for TB It's gonna Joe Max Kepler for the twins We're in a path. All right, then church is gonna be my little my blank Good luck. We have a framed autograph card of Damn, it's Aaron Hicks Tyler. You're correct Yankees going to Brian Crouch All right, let's go to the next eight. You win a good job Virtual high five You got to fill it up those love is $50 and $60 team so let's go All right, good luck us Gavin Luxe Louise Roberts Looks again trout class three Your dawn Shit Tatis tonight and I ain't got Ricky Henderson relic for the A's when a Jarell Yoshi All right guys Yoshi Three more that will go through all the autos guys 33 minutes 75 Thank her job You go Well tiny 36 out of 50 last base one right here, and then we'll go through all the autos Chris Bryant to 25 This is the Zardo to 25 and Louise Robert Carlos All right, I guess good luck Justin Dunn to start us off for Seattle When a DMAC Bobby Bradley for Cleveland Gonna Mike Michael Brucelle For TV and a Randy Arosa reina. There you go Joe with the Tampa Bay Rays I feel like he's gonna be a more common auto in most of these products now because he's kind of just exploded These last couple of weeks with the playoffs But in products Maybe kind of normal Patrick Corbin 51 out of 75 Washington going to Robert Mancini. I threw more guys. Good luck Jake Rogers for Detroit Brown out that one I have two more guys Junior Fernandez All right Last one guys. Good luck Just casting right there class three Wow and a Mike Schmidt Two out of 25 little relic bat relic and auto For the Phillies Glen Howard as a custom class three actually And there you go guys that was the break all really nice hits here. I love these frames And again a really solid price point for the product Got a Ronald Acunia Very very nice appreciate it guys. This was tops gold label baseball 16 bucks case for a quicker teams number eight on Jaspies case breaks


that was just a candle maker shot. Franklin, reading other good writers, became a writer. And one of the scores, so throughout the ACP, and again, sometimes under student M, sometimes under his own process of setting the type for the deadly disease that would often sweep. But in his home in Philadelphia, that they disagreed with some of the Penn family's policies, particularly regarding taxation. So he's there in London, you know, stamps for every little thing. That, he thought, was the province of. Franklin was given some letters by Michael, that one of the crowd shot him. And this is a printed copy negotiated.


So there are two ways that we can grow our bone and cartilage tissue. And if you think about it, it makes sense that you would have a special type or a way, a mechanism by which growth takes place because we're dealing with a solid, a relatively solid tissue. And so it's a little bit funky. So first of all, we can actually have a process of oppositional growth and this takes place on the surface. So let's just make a little note. This is like on the surface. And interstitial growth is like in the middle. Now here's the deal. Both of these types of growth occur in cartilage and bone. However, they're slightly different. Now, oppositional growth, does it make sense? It takes place on the surface. So basically what happens is at the edge of your tissue, near, let's say, your pericondrium or periosteum, basically you're just going to have cell division take place. And we're going to end up with new cells. And those new cells are going to grow, whether it be chondroblasts or osteoblasts, they're going to barf out more matrix around them. And then the bone is going to increase in diameter or the cartilage is going to increase in diameter. So the growth is going to happen this direction. Can you visualize that? This is increase in diameter. And that happens through oppositional growth. Now, interstitial growth happens in cartilage. It makes complete sense. So the lacuna, you have a cell inside the lacuna and then something triggers, it's time to grow. And so the cell will divide and become two cells. And then the cell, which was an osteocyte, right, because it was just chilling in its lacuna, excuse me, it's a chondrocyte, because this exact form doesn't exactly happen this way in bone. But the chondrocyte is going to go back and become a chondroblast again. So some kind of chemical message gets sent when interstitial growth is going to take place. And now these guys are going to start barfing out their matrix. And basically, they're going to condense the tissue. Like, they're going to squeeze more. They're going to keep their lacuna, but they squish out more matrix around them, so the matrix becomes more dense. Does that, I mean, I could totally visualize that. It's like, if I were in this room and then I wanted to, and the walls were made out of clay, and I wanted to, you know, make more clay, I could totally push more clay into the walls and the walls would become more dense. And the clay analogy isn't fantastic. Something that squishes down becomes dense, like Wonder Bread. So I'm going to squish more Wonder Bread onto my walls, and then, like, they started out being all Wonder Bread-y, but then I can squish more. Like, I could probably fit an entire loaf of Wonder Bread where I had one piece of Wonder Bread, because I could squish it down. That's what your matrix in cartilage is like that. So your matrix just becomes more dense. Your tissue becomes more dense with interstitial growth in the cartilage tissue. Interstitial growth. In compact bone, can you visualize what interstitial growth happened? You can't make bone tissue compact-er. You can't squish it more. It's already solid. However, thank you very much. I would like to say interstitial growth does not happen in bone. Unfortunately, they use the same word for a slightly different process. They say growth at an epiphyseal plate. Bone growth increased length at an epiphyseal plate. They call that interstitial. But think about it for a second. Was the epiphyseal plate? It was hyaline cartilage. That's not bone. So sure, the hyaline cartilage can expand and can create more bone tissue. And we'll look at that whole process in lab today or whenever you come into lab. But that is a different... It's hyaline cartilage that's growing and turning into more bone. However, they refer to it as interstitial bone growth. So, okay, that's cool. Do you have any questions for me? All right. I think that's it, except for our histology preview, which might have to happen in a second.